Document Detail


Immune exhaustion occurs concomitantly with immune activation and decrease in regulatory T cells in viremic chronically HIV-1-infected patients.
MedLine Citation:
PMID:  20463584     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: Chronic HIV-1 infection is associated with excessive immune activation and immune exhaustion. We investigated the relationship of these 2 phenotypes and frequency of regulatory T cells (Tregs) in controlled and uncontrolled chronic HIV-1 infection.
METHODS: Immune exhaustion marker PD-1, its ligand PD-L1, CD4CD25 FoxP3 Tregs, HLA-DR, and CD38 coexpression as activation markers were investigated in peripheral blood lymphocytes of 44 HIV-1-infected patients and 11 HIV-1-uninfected controls by multicolor flow cytometry.
RESULTS: Activated and PD-1 expressing T cells were increased, and Tregs were decreased in HIV-1-infected patients as compared with controls, and alterations were greatest in viremic patients. The proportion of activated CD8 T cells exceeded activated CD4 T cells. Tregs had an inverse correlation with activated T cells and PD-1 expressing T cells. PD-L1 was highly expressed on monocytes and to a lesser extent on T lymphocytes of patients. These abnormalities partially reversed with virologic control after potent antiretroviral therapy.
CONCLUSIONS: Immune exhaustion is a component of aberrant immune activation in chronic HIV-1 infection and is associated with loss of Tregs and ongoing virus replication. These defects are corrected partially with effective virologic control by potent antiretroviral therapy.
Authors:
Meenakshi Sachdeva; Margaret A Fischl; Rajendra Pahwa; Naresh Sachdeva; Savita Pahwa
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of acquired immune deficiency syndromes (1999)     Volume:  54     ISSN:  1944-7884     ISO Abbreviation:  J. Acquir. Immune Defic. Syndr.     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-07-28     Completed Date:  2010-08-12     Revised Date:  2011-09-06    
Medline Journal Info:
Nlm Unique ID:  100892005     Medline TA:  J Acquir Immune Defic Syndr     Country:  United States    
Other Details:
Languages:  eng     Pagination:  447-54     Citation Subset:  IM; X    
Affiliation:
Department of Microbiology and Immunology, Developmental Center for AIDS Research, University of Miami-Miller School of Medicine, Miami, FL 33136, USA.
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MeSH Terms
Descriptor/Qualifier:
Adult
Antigens, CD / analysis
Chronic Disease
Female
Flow Cytometry
HIV Infections / immunology*
HIV-1 / immunology*
Humans
Lymphocyte Activation*
Male
T-Lymphocyte Subsets / chemistry,  immunology
T-Lymphocytes, Regulatory / immunology*
Grant Support
ID/Acronym/Agency:
AI068632/AI/NIAID NIH HHS; R01 AI077501-01/AI/NIAID NIH HHS; U01 AI068632/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Antigens, CD
Comments/Corrections

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