Document Detail

Hypoxic-ischemic encephalopathy in the newborn.
MedLine Citation:
PMID:  6405725     Owner:  NLM     Status:  MEDLINE    
Many term newborns suffer some degree of perinatal asphyxia, but few become permanently brain damaged as a consequence, The newborns at risk for major neurologic handicaps have evidence of derangement in many organs, depressed cerebral function at birth that continues for days or weeks, and in many cases, convulsions soon after birth. The pathophysiologic mechanism of brain damage in hypoxic-ischemic encephalopathy remains contested, and no specific therapy is clearly beneficial. The immediate treatment of the asphyxiated term newborn requires attention to the derangements in multiple organ systems and the use of anticonvulsants when indicated.
G M Fenichel
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Archives of neurology     Volume:  40     ISSN:  0003-9942     ISO Abbreviation:  Arch. Neurol.     Publication Date:  1983 May 
Date Detail:
Created Date:  1983-06-10     Completed Date:  1983-06-10     Revised Date:  2004-11-17    
Medline Journal Info:
Nlm Unique ID:  0372436     Medline TA:  Arch Neurol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  261-6     Citation Subset:  AIM; IM    
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MeSH Terms
Asphyxia Neonatorum / diagnosis,  drug therapy,  metabolism,  physiopathology*
Brain Ischemia / diagnosis,  drug therapy,  metabolism,  physiopathology*
Cerebrovascular Circulation
Disease Models, Animal
Energy Metabolism
Glucose / metabolism
Hypoxia, Brain / diagnosis,  drug therapy,  metabolism,  physiopathology*
Infant, Newborn
Infant, Newborn, Diseases / diagnosis,  drug therapy,  metabolism,  physiopathology
Lactates / metabolism
Reg. No./Substance:
0/Lactates; 50-99-7/Glucose

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