| Hypothetical pathways of cardiac myocyte hypertrophy: response to myocardial injury. | |
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MedLine Citation:
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PMID: 8281963 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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The heart is made up of many different cell types, including myocytes, fibroblasts and vascular cells. The cardiac myocyte, although not likely to divide, may respond to gross organ injury by a process of growth or hypertrophy. Cellular hypertrophy, though a normal biological adaptation, has the potential to contribute to the change in the shape and the size of the heart, such as occurs in chronic heart failure. We are now just beginning to understand the signals and early pathways involved in the cardiac myocyte growth response, some of which are reviewed herein. |
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Authors:
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G S Francis; W C Carlyle |
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Publication Detail:
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Type: Journal Article; Review |
Journal Detail:
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Title: European heart journal Volume: 14 Suppl J ISSN: 0195-668X ISO Abbreviation: Eur. Heart J. Publication Date: 1993 Nov |
Date Detail:
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Created Date: 1994-02-14 Completed Date: 1994-02-14 Revised Date: 2005-11-16 |
Medline Journal Info:
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Nlm Unique ID: 8006263 Medline TA: Eur Heart J Country: ENGLAND |
Other Details:
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Languages: eng Pagination: 49-56 Citation Subset: IM |
Affiliation:
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Department of Medicine, University of Minnesota Medical School, Minneapolis 55455. |
Export Citation:
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| MeSH Terms | |
Descriptor/Qualifier:
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Animals Cardiomegaly / physiopathology* Cell Division Humans Hypertrophy Myocardial Infarction / pathology*, physiopathology Myocardium / cytology* Signal Transduction |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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