Document Detail


Hypokalemic paralysis due to Gitelman syndrome: a family study.
MedLine Citation:
PMID:  17000984     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hypokalemic paralysis is rarely seen as the presenting feature in patients with Gitelman syndrome. We report a Chinese man who presented with periodic paralysis, in whom molecular analysis revealed compound heterozygous inheritance of three mutations of the thiazide-sensitive sodium chloride cotransporter. Family history revealed intrafamilial variation in phenotypes. Gitelman syndrome should be considered as a cause of hypokalemic paralysis, and molecular analysis may help establish the diagnosis.
Authors:
H-Y Ng; S-H Lin; C-Y Hsu; Y-Z Tsai; H-C Chen; C-T Lee
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Publication Detail:
Type:  Case Reports; Comparative Study; Journal Article    
Journal Detail:
Title:  Neurology     Volume:  67     ISSN:  1526-632X     ISO Abbreviation:  Neurology     Publication Date:  2006 Sep 
Date Detail:
Created Date:  2006-09-26     Completed Date:  2006-10-11     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0401060     Medline TA:  Neurology     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1080-2     Citation Subset:  AIM; IM    
Affiliation:
Division of Nephrology, Department of Medicine, Chang-Gung Memorial Hospital, Kaohsiung, Taiwan.
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MeSH Terms
Descriptor/Qualifier:
Adult
Exons
Family Health*
Female
Humans
Hypokalemia* / complications,  diagnosis,  genetics
Hypokalemic Periodic Paralysis / diagnosis,  etiology*,  genetics
Kidney Diseases / complications*,  genetics
Male
Mutation
Sodium-Potassium-Chloride Symporters / genetics
Syndrome
Chemical
Reg. No./Substance:
0/Sodium-Potassium-Chloride Symporters; 0/sodium-potassium chloride cotransporter 2 protein

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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