Document Detail


Hypersociability in Williams syndrome: a role for the amygdala?
MedLine Citation:
PMID:  18622788     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We read with great interest the paper of Riby et al. regarding atypical, unfamiliar face processing in Williams syndrome (WS; Riby, Doherty-Sneddon, & Bruce, 2008a). It offers considerable insight into the mechanism of facial perception in humans and a further elaboration of the hypersociability observed in patients with Williams syndrome. We would like to suggest that the neurologic mechanisms underlying the hypersociability in WS may be attributable to an impaired recognition of facial expressions of threat, a feature that localises to the amygdala.
Authors:
Ali Jawaid; Heike Schmolck; Paul E Schulz
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Publication Detail:
Type:  Comment; Journal Article    
Journal Detail:
Title:  Cognitive neuropsychiatry     Volume:  13     ISSN:  1464-0619     ISO Abbreviation:  Cogn Neuropsychiatry     Publication Date:  2008 Jul 
Date Detail:
Created Date:  2008-07-14     Completed Date:  2008-09-25     Revised Date:  2009-01-16    
Medline Journal Info:
Nlm Unique ID:  9713497     Medline TA:  Cogn Neuropsychiatry     Country:  England    
Other Details:
Languages:  eng     Pagination:  338-42     Citation Subset:  IM    
Affiliation:
Department of Neurology, Baylor College of Medicine, Houston, TX 77030, USA.
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MeSH Terms
Descriptor/Qualifier:
Adolescent
Amygdala / physiopathology*
Child
Dominance, Cerebral / physiology
Emotions / physiology*
Facial Expression*
Female
Humans
Judgment
Male
Perceptual Disorders / physiopathology*,  psychology
Personal Construct Theory
Social Behavior*
Visual Perception / physiology*
Williams Syndrome / physiopathology*,  psychology
Comments/Corrections
Comment On:
Cogn Neuropsychiatry. 2008 Jan;13(1):47-58   [PMID:  18092225 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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