Document Detail


Hyperoxia-derived lung damage in preterm infants.
MedLine Citation:
PMID:  20430708     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hyperoxia-induced lung injury is characterized by an influx of inflammatory cells, increased pulmonary permeability, endothelial and epithelial cell death. This review highlights the mechanistic aspects of inflammation, vascular leak and cell death. The focus will be on agents that contribute to hyperoxia-induced lung injury in developmentally appropriate animal models, and those that have been detected in human premature neonates.
Authors:
Vineet Bhandari
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-04-28
Journal Detail:
Title:  Seminars in fetal & neonatal medicine     Volume:  15     ISSN:  1878-0946     ISO Abbreviation:  Semin Fetal Neonatal Med     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-07-26     Completed Date:  2010-11-15     Revised Date:  2014-09-13    
Medline Journal Info:
Nlm Unique ID:  101240003     Medline TA:  Semin Fetal Neonatal Med     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  223-9     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Ltd. All rights reserved.
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MeSH Terms
Descriptor/Qualifier:
Animals
Disease Models, Animal
Humans
Hyperoxia / metabolism,  pathology*
Infant, Newborn
Infant, Premature / metabolism*
Lung Injury / metabolism,  pathology*
Grant Support
ID/Acronym/Agency:
HL-074195/HL/NHLBI NIH HHS; HL-085103/HL/NHLBI NIH HHS; K08 HL074195/HL/NHLBI NIH HHS; K08 HL074195-05/HL/NHLBI NIH HHS; R01 HL085103/HL/NHLBI NIH HHS; R01 HL085103-01A2/HL/NHLBI NIH HHS
Comments/Corrections

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