Document Detail


Hyper IgM syndrome and complement Clq deficiency in an individual with systemic lupus erythematosus-like disease.
MedLine Citation:
PMID:  20810037     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Many immunedeficiency syndromes are associated with autoimmune disorders. We here report on a girl with a systemic lupus erythematosus-like disease who suffered from both hyperimmunoglobulin M syndrome (HIGMS) and C1q deficiency. Despite severe central nervous system-lupus like disease, probably due to C1q deficiency, kidney function was relatively spared. IgM autoantibody might play a protective role against lupus-glomerulonephritis.
Authors:
I Tsuge; Y Kondo; Y Nakajima; N Nakagawa; K Imai; S Nonoyama; K Oshima; O Ohara; M Hatanaka; E Kitano; H Kitamura; A Urisu
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Publication Detail:
Type:  Case Reports; Journal Article     Date:  2010-08-30
Journal Detail:
Title:  Clinical and experimental rheumatology     Volume:  28     ISSN:  0392-856X     ISO Abbreviation:  Clin. Exp. Rheumatol.     Publication Date:    2010 Jul-Aug
Date Detail:
Created Date:  2010-09-02     Completed Date:  2010-12-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8308521     Medline TA:  Clin Exp Rheumatol     Country:  Italy    
Other Details:
Languages:  eng     Pagination:  558-60     Citation Subset:  IM    
Affiliation:
Department of Paediatrics, Fujita Health University, Aichi, Japan. itsuge@fujita-hu.ac.jp
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MeSH Terms
Descriptor/Qualifier:
Child
Complement C1q / deficiency*
Female
Humans
Hyper-IgM Immunodeficiency Syndrome / complications*,  diagnosis
Immunoglobulin M / physiology
Lupus Erythematosus, Systemic / complications,  diagnosis,  etiology*
Lupus Nephritis / physiopathology,  prevention & control
Chemical
Reg. No./Substance:
0/Immunoglobulin M; 80295-33-6/Complement C1q

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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