Document Detail


Hydrogen sulfide facilitates carotid sinus baroreflex in anesthetized rats.
MedLine Citation:
PMID:  16490164     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
AIM: To study effects of hydrogen sulfide (H2S) on the carotid sinus baroreflex (CSB). METHODS: The functional curve of the carotid sinus baroreflex was measured by recording changes in arterial pressure in anesthetized male rats with perfused carotid sinus. RESULTS: H2S (derived from sodium hydrosulfide) at concentrations of 25, 50, and 100 micromol/L facilitated the CSB, shifting the functional curve of the baroreflex downward and to the left. There was a marked increase in peak slope (PS) and reflex decrease in blood pressure (RD). Effects were concentration-dependent. Pretreatment with glibenclamide (20 micromol/L), a K(ATP) channel blocker, abolished the above effects of H2S on CSB. Pretreatment with Bay K8644 (an agonist of calcium channels; 500 nmol/L) eliminated the effect of H2S on CSB. An inhibitor of cystathionine gamma-lyase (CSE), DL-propargylglycine (PPG; 200 micromol/L), inhibited CSB in male rats and shifted the functional curve of the baroreflex upward and to the right. CONCLUSION: These data suggest that exogenous H2S exerts a facilitatory role on isolated CSB through opening K(ATP) channels and further closing the calcium channels in vascular smooth muscle. Endogenous H2S may activate the activity of the CSB in vivo.
Authors:
Lin Xiao; Yu-ming Wu; Hao Zhang; Yi-xian Liu; Rui-rong He
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Acta pharmacologica Sinica     Volume:  27     ISSN:  1671-4083     ISO Abbreviation:  Acta Pharmacol. Sin.     Publication Date:  2006 Mar 
Date Detail:
Created Date:  2006-02-21     Completed Date:  2006-10-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100956087     Medline TA:  Acta Pharmacol Sin     Country:  China    
Other Details:
Languages:  eng     Pagination:  294-8     Citation Subset:  IM    
Affiliation:
Department of Physiology, Institute of Basic Medicine, Hebei Medical University, Shijiazhuang 050017, China.
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MeSH Terms
Descriptor/Qualifier:
3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester / pharmacology
Alkynes / pharmacology
Animals
Baroreflex / drug effects*
Blood Pressure / drug effects
Calcium Channel Agonists / pharmacology
Carotid Sinus / drug effects,  physiology*
Cystathionine gamma-Lyase / antagonists & inhibitors
Glyburide / pharmacology
Glycine / analogs & derivatives,  pharmacology
Hydrogen Sulfide / pharmacology*
Male
Potassium Channel Blockers / pharmacology
Rats
Rats, Sprague-Dawley
Sulfides / pharmacology
Chemical
Reg. No./Substance:
0/Alkynes; 0/Calcium Channel Agonists; 0/Potassium Channel Blockers; 0/Sulfides; 10238-21-8/Glyburide; 16721-80-5/sodium bisulfide; 56-40-6/Glycine; 64165-64-6/propargylglycine; 71145-03-4/3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; 7783-06-4/Hydrogen Sulfide; EC 4.4.1.1/Cystathionine gamma-Lyase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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