Document Detail


Human transferrin confers serum resistance against Bacillus anthracis.
MedLine Citation:
PMID:  20615872     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The innate immune system in humans consists of both cellular and humoral components that collaborate to eradicate invading bacteria from the body. Here, we discover that the gram-positive bacterium Bacillus anthracis, the causative agent of anthrax, does not grow in human serum. Fractionation of serum by gel filtration chromatography led to the identification of human transferrin as the inhibiting factor. Purified transferrin blocks growth of both the fully virulent encapsulated B. anthracis Ames and the non-encapsulated Sterne strain. Growth inhibition was also observed in serum of wild-type mice but not of hypotransferrinemic mice that only have approximately 1% circulating transferrin levels. We were able to definitely assign the bacteriostatic activity of transferrin to its iron-binding function: neither iron-saturated transferrin nor a recombinant transferrin mutant unable to bind iron could inhibit growth of B. anthracis. Additional iron could restore bacterial growth in human serum. The observation that other important gram-positive pathogens are not inhibited by transferrin suggests they have evolved effective mechanisms to circumvent serum iron deprivation. These findings provide a better understanding of human host defense mechanisms against anthrax and provide a mechanistic basis for the antimicrobial activity of human transferrin.
Authors:
Suzan H M Rooijakkers; Suzanne L Rasmussen; Shauna M McGillivray; Thomas B Bartnikas; Anne B Mason; Arthur M Friedlander; Victor Nizet
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-07-08
Journal Detail:
Title:  The Journal of biological chemistry     Volume:  285     ISSN:  1083-351X     ISO Abbreviation:  J. Biol. Chem.     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-08-30     Completed Date:  2010-10-05     Revised Date:  2011-09-13    
Medline Journal Info:
Nlm Unique ID:  2985121R     Medline TA:  J Biol Chem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  27609-13     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, University of California, San Diego, La Jolla, California 92093, USA. s.h.m.rooijakkers@umcutrecht.nl
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MeSH Terms
Descriptor/Qualifier:
Animals
Anti-Bacterial Agents / pharmacology*
Bacillus anthracis / drug effects*,  growth & development
Female
Humans
Immunity, Innate
Iron / metabolism
Mice
Serum / immunology*,  microbiology*
Transferrin / pharmacology*
Grant Support
ID/Acronym/Agency:
AI077780/AI/NIAID NIH HHS; DK084122-01/DK/NIDDK NIH HHS; DK21739/DK/NIDDK NIH HHS; GM068524/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/Anti-Bacterial Agents; 11096-37-0/Transferrin; 7439-89-6/Iron
Comments/Corrections

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