Document Detail


Human retinal pigment epithelium-induced CD4+CD25+ regulatory T cells suppress activation of intraocular effector T cells.
MedLine Citation:
PMID:  20350837     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Murine retinal pigment epithelial (RPE) cells suppress T-cell activation by releasing soluble inhibitory factors and promote the generation of regulatory T cells in vitro. These T cells exposed to RPE supernatants (RPE-induced Treg cells) can suppress the activation of bystander effector T cells via the production of transforming growth factor-beta (TGFbeta). In the present study, we showed that human RPE-induced Treg cells are also able to acquire regulatory function when human RPE cell lines were pretreated with recombinant TGF beta 2. These RPE-induced Treg cells produced TGF beta 1 and IL-10 but not IFN gamma, and they significantly suppressed the activation of target cell lines and intraocular T-cell clones established from patients with active uveitis. Moreover, CD4(+)CD25(+) RPE-induced Treg cells expressed CTLA-4 and Foxp3 molecules, and the CD25(+) Treg cells profoundly suppressed the T-cell activation. Thus, in vitro manipulated Treg cells acquire functions that participate in the establishment of immune tolerance in the eye.
Authors:
Shintaro Horie; Sunao Sugita; Yuri Futagami; Yukiko Yamada; Manabu Mochizuki
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-03-29
Journal Detail:
Title:  Clinical immunology (Orlando, Fla.)     Volume:  136     ISSN:  1521-7035     ISO Abbreviation:  Clin. Immunol.     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-06-23     Completed Date:  2010-08-06     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100883537     Medline TA:  Clin Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  83-95     Citation Subset:  IM    
Copyright Information:
(c) 2010 Elsevier Inc. All rights reserved.
Affiliation:
Department of Ophthalmology & Visual Science, Tokyo Medical and Dental University, Graduate School of Medical and Dental Sciences, Tokyo, Japan.
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MeSH Terms
Descriptor/Qualifier:
Antibodies, Monoclonal / immunology,  pharmacology
CD4-Positive T-Lymphocytes / immunology,  metabolism
Cell Line
Cell Proliferation
Culture Media, Conditioned / pharmacology
Dinoprostone / metabolism
Epithelial Cells / drug effects,  immunology,  metabolism
Eye / cytology,  immunology*
Forkhead Transcription Factors / metabolism
Gene Expression / drug effects,  genetics
Humans
Immune Tolerance / immunology*
Interferon-gamma / metabolism
Interleukin-10 / metabolism
Interleukin-2 Receptor alpha Subunit / metabolism*
Lymphocyte Activation / immunology
RNA, Small Interfering / genetics
Receptors, Transforming Growth Factor beta / genetics
Retinal Pigment Epithelium / cytology,  immunology*
T-Lymphocytes / immunology*,  metabolism
T-Lymphocytes, Regulatory / drug effects,  immunology*,  metabolism
Th1 Cells / immunology,  metabolism
Thrombospondin 1 / metabolism
Transforming Growth Factor beta / antagonists & inhibitors,  immunology
Transforming Growth Factor beta1 / genetics,  metabolism
Transforming Growth Factor beta2 / genetics,  metabolism,  pharmacology
Uveitis / immunology
Chemical
Reg. No./Substance:
0/Antibodies, Monoclonal; 0/Culture Media, Conditioned; 0/FOXP3 protein, human; 0/Forkhead Transcription Factors; 0/IL10 protein, human; 0/IL2RA protein, human; 0/Interleukin-2 Receptor alpha Subunit; 0/RNA, Small Interfering; 0/Receptors, Transforming Growth Factor beta; 0/Thrombospondin 1; 0/Transforming Growth Factor beta; 0/Transforming Growth Factor beta1; 0/Transforming Growth Factor beta2; 130068-27-8/Interleukin-10; 363-24-6/Dinoprostone; 82115-62-6/Interferon-gamma

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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