Document Detail


Human gingiva-derived mesenchymal stem cells elicit polarization of m2 macrophages and enhance cutaneous wound healing.
MedLine Citation:
PMID:  20734355     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Increasing evidence has supported the important role of mesenchymal stem cells (MSCs) in wound healing, however, the underlying mechanism remains unclear. Recently, we have isolated a unique population of MSCs from human gingiva (GMSCs) with similar stem cell-like properties, immunosuppressive, and anti-inflammatory functions as human bone marrow-derived MSCs (BMSCs). We describe here the interplay between GMSCs and macrophages and the potential relevance in skin wound healing. When cocultured with GMSCs, macrophages acquired an anti-inflammatory M2 phenotype characterized by an increased expression of mannose receptor (MR; CD206) and secretory cytokines interleukin (IL)-10 and IL-6, a suppressed production of tumor necrosis factor (TNF)-α, and decreased ability to induce Th-17 cell expansion. In vivo, we demonstrated that systemically infused GMSCs could home to the wound site in a tight spatial interaction with host macrophages, promoted them toward M2 polarization, and significantly enhanced wound repair. Mechanistically, GMSC treatment mitigated local inflammation mediated by a suppressed infiltration of inflammatory cells and production of IL-6 and TNF-α, and an increased expression of IL-10. The GMSC-induced suppression of TNF-α secretion by macrophages appears to correlate with impaired activation of NFκB p50. These findings provide first evidence that GMSCs are capable to elicit M2 polarization of macrophages, which might contribute to a marked acceleration of wound healing.
Authors:
Qun-Zhou Zhang; Wen-Ru Su; Shi-Hong Shi; Petra Wilder-Smith; Andy Peng Xiang; Alex Wong; Andrew L Nguyen; Chan Wook Kwon; Anh D Le
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Stem cells (Dayton, Ohio)     Volume:  28     ISSN:  1549-4918     ISO Abbreviation:  Stem Cells     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-10-27     Completed Date:  2011-02-08     Revised Date:  2012-04-13    
Medline Journal Info:
Nlm Unique ID:  9304532     Medline TA:  Stem Cells     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1856-68     Citation Subset:  IM    
Affiliation:
Center for Craniofacial Molecular Biology, The Herman Ostrow School of Dentistry of University of Southern California, Los Angeles, California 90033, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Blotting, Western
Cell Line
Cells, Cultured
Coculture Techniques
Enzyme-Linked Immunosorbent Assay
Flow Cytometry
Gingiva / cytology*
Humans
Macrophages / cytology*,  immunology*
Male
Mesenchymal Stem Cells / cytology*,  metabolism*
Mice
Mice, Inbred C57BL
Wound Healing / immunology*
Grant Support
ID/Acronym/Agency:
R01 DE019932-02/DE/NIDCR NIH HHS; R01 DE019932-04/DE/NIDCR NIH HHS; R01DE 019932/DE/NIDCR NIH HHS
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