| Host interception of bacterial communication signals. | |
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MedLine Citation:
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PMID: 19064249 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Pathogenic Staphylococcus aureus employs a cell-to-cell communication system to control virulence factor expression in the host during infection. In this issue, Peterson et al. (2008) report that host lipoproteins can sequester the S. aureus communication signal and antagonize virulence factor expression and pathogenesis in a murine model of infection. |
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Authors:
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Alexander R Horswill; William M Nauseef |
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Publication Detail:
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Type: Comment; Journal Article |
Journal Detail:
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Title: Cell host & microbe Volume: 4 ISSN: 1934-6069 ISO Abbreviation: Cell Host Microbe Publication Date: 2008 Dec |
Date Detail:
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Created Date: 2008-12-09 Completed Date: 2009-01-02 Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 101302316 Medline TA: Cell Host Microbe Country: United States |
Other Details:
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Languages: eng Pagination: 507-9 Citation Subset: IM |
Affiliation:
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Department of Microbiology, Roy J and Lucille A Carver College of Medicine, University of Iowa, Iowa City, IA 52242, USA. alex-horswill@uiowa.edu |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Animals Bacterial Proteins / antagonists & inhibitors* Lipoproteins / immunology* Mice Staphylococcal Infections / immunology* Staphylococcus aureus / pathogenicity* |
| Chemical | |
Reg. No./Substance:
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0/AgrD protein, Staphylococcus; 0/Bacterial Proteins; 0/Lipoproteins |
| Comments/Corrections | |
Comment On:
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Cell Host Microbe. 2008 Dec 11;4(6):555-66
[PMID:
19064256
]
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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