| Histochemical findings of the tribocytic organ and tegument of Fibricola seoulensis. | |
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MedLine Citation:
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PMID: 2151308 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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The tribocytic organ and tegument of Fibricola seoulensis were examined histochemically for the detection of carbohydrates, mucosubstances, amyloid, collagen and alkaline phosphatase. The surface, secretes, gland cells of the tribocytic organ, and the tegument of the worms were positive to periodic acid Schiff (PAS) and PAS with diastase stain but negative to other stains. It was inferred that the tribocytic organ and tegument of F. seoulensis comprise neutral mucopolysaccharides, which may take a protective role against host enzymes. The surface and secretes of the tribocytic organ, and the tegument of the worms were also positive to double bridge PAP for alkaline phosphatase. This fact suggests that they may play a role as both self protective and host tissue lytic functions. |
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Authors:
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S Huh; S H Lee; B S Seo |
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Publication Detail:
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Type: Journal Article; Research Support, Non-U.S. Gov't |
Journal Detail:
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Title: Kisaengch'unghak chapchi. The Korean journal of parasitology Volume: 28 ISSN: - ISO Abbreviation: Kisaengchunghak Chapchi Publication Date: 1990 Sep |
Date Detail:
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Created Date: 1991-07-08 Completed Date: 1991-07-08 Revised Date: 2006-11-15 |
Medline Journal Info:
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Nlm Unique ID: 0366132 Medline TA: Kisaengchunghak Chapchi Country: KOREA |
Other Details:
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Languages: eng Pagination: 155-60 Citation Subset: IM |
Affiliation:
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Department of Parasitology, College of Medicine, Hallym University, Chunchon, Korea. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Alkaline Phosphatase
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analysis*,
physiology Amyloid / analysis Animals Collagen / analysis Glycosaminoglycans / analysis*, physiology Histocytochemistry Mice Trematoda / cytology, metabolism*, pathogenicity |
| Chemical | |
Reg. No./Substance:
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0/Amyloid; 0/Glycosaminoglycans; 9007-34-5/Collagen; EC 3.1.3.1/Alkaline Phosphatase |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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