Document Detail


Hippocampal memory processes are modulated by insulin and high-fat-induced insulin resistance.
MedLine Citation:
PMID:  20176121     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Insulin regulates glucose uptake and storage in peripheral tissues, and has been shown to act within the hypothalamus to acutely regulate food intake and metabolism. The machinery for transduction of insulin signaling is also present in other brain areas, particularly in the hippocampus, but a physiological role for brain insulin outside the hypothalamus has not been established. Recent studies suggest that insulin may be able to modulate cognitive functions including memory. Here we report that local delivery of insulin to the rat hippocampus enhances spatial memory, in a PI-3-kinase dependent manner, and that intrahippocampal insulin also increases local glycolytic metabolism. Selective blockade of endogenous intrahippocampal insulin signaling impairs memory performance. Further, a rodent model of type 2 diabetes mellitus produced by a high-fat diet impairs basal cognitive function and attenuates both cognitive and metabolic responses to hippocampal insulin administration. Our data demonstrate that insulin is required for optimal hippocampal memory processing. Insulin resistance within the telencephalon may underlie the cognitive deficits commonly reported to accompany type 2 diabetes.
Authors:
Ewan C McNay; Cecilia T Ong; Rory J McCrimmon; James Cresswell; Jonathan S Bogan; Robert S Sherwin
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-02-20
Journal Detail:
Title:  Neurobiology of learning and memory     Volume:  93     ISSN:  1095-9564     ISO Abbreviation:  Neurobiol Learn Mem     Publication Date:  2010 May 
Date Detail:
Created Date:  2010-05-03     Completed Date:  2010-07-30     Revised Date:  2011-09-19    
Medline Journal Info:
Nlm Unique ID:  9508166     Medline TA:  Neurobiol Learn Mem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  546-53     Citation Subset:  IM    
Copyright Information:
Published by Elsevier Inc.
Affiliation:
Dept. of Psychology, University at Albany, Albany, NY 12222, USA. emcnay@albany.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Diabetes Mellitus, Experimental / metabolism
Diet
Dietary Fats / metabolism
Disease Models, Animal
Hippocampus / drug effects,  enzymology,  physiology*
Insulin / metabolism*
Insulin Resistance / physiology*
Male
Memory / drug effects,  physiology*
Memory Disorders / metabolism
Phosphatidylinositol 3-Kinases / metabolism
Random Allocation
Rats
Rats, Sprague-Dawley
Space Perception / drug effects,  physiology
Grant Support
ID/Acronym/Agency:
DK 45735/DK/NIDDK NIH HHS; DK075772/DK/NIDDK NIH HHS; DK077106/DK/NIDDK NIH HHS; R01 DK075772-02/DK/NIDDK NIH HHS; R01 DK075772-03/DK/NIDDK NIH HHS; R01 DK075772-05/DK/NIDDK NIH HHS; R01 DK077106-01A1/DK/NIDDK NIH HHS; R01 DK077106-02/DK/NIDDK NIH HHS; R01 DK077106-03/DK/NIDDK NIH HHS; R01 DK077106-04/DK/NIDDK NIH HHS; R01 DK077106-05/DK/NIDDK NIH HHS; R37 DK020495-35/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Dietary Fats; 11061-68-0/Insulin; EC 2.7.1.-/Phosphatidylinositol 3-Kinases
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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