Document Detail


High risk for neoplastic transformation of endometriosis in a carrier of Lynch syndrome.
MedLine Citation:
PMID:  20087665     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Lynch syndrome is an autosomal dominant cancer-susceptibility disorder caused by mutations in DNA mismatch repair genes. Women with Lynch syndrome have an increased lifetime risk for endometrial and ovarian cancers. While there is evidence of efficacy for prophylactic surgery, no standard recommendations have been developed to support screening for premalignant endometrial and ovarian epithelial lesions in high-risk women. Here, we report a case of a healthy woman carrying a germline mutation in MLH1 gene with endometrial intra-epithelial neoplasia and ovarian endometriotic lesions exhibiting a loss of MLH1 protein expression. This case report illustrates the malignant potential of endometriosis, and highlights the need for a meticulous management of gynecologic premalignant precursor lesions in reducing cancer risk among related Lynch syndrome women.
Authors:
Christine Nyiraneza; Etienne Marbaix; Mireille Smets; Christine Galant; Christine Sempoux; Karin Dahan
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Publication Detail:
Type:  Case Reports; Journal Article    
Journal Detail:
Title:  Familial cancer     Volume:  9     ISSN:  1573-7292     ISO Abbreviation:  Fam. Cancer     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-08-16     Completed Date:  2010-12-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100898211     Medline TA:  Fam Cancer     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  383-7     Citation Subset:  IM    
Affiliation:
Center for Human Genetics, Cliniques universitaires Saint-Luc, Université catholique de Louvain, Brussels, Belgium.
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MeSH Terms
Descriptor/Qualifier:
Adaptor Proteins, Signal Transducing / genetics
Carcinoma in Situ / genetics,  pathology*
Cell Transformation, Neoplastic / genetics*
Colorectal Neoplasms, Hereditary Nonpolyposis / genetics,  pathology*
Endometrial Neoplasms / genetics,  pathology*
Endometriosis / genetics,  pathology*
Female
Genetic Predisposition to Disease
Heterozygote
Humans
Middle Aged
Mutation
Nuclear Proteins / genetics
Pedigree
Risk Factors
Chemical
Reg. No./Substance:
0/Adaptor Proteins, Signal Transducing; 0/MLH1 protein, human; 0/Nuclear Proteins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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