Document Detail


High-resolution magic-angle-spinning 1H NMR spectroscopy reveals different responses in choline-containing metabolites upon gene therapy-induced programmed cell death in rat brain glioma.
MedLine Citation:
PMID:  15884096     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Changes in the concentrations of choline-containing metabolites (CCM) have been implicated in both cell proliferation and death processes. In this study, high-resolution magic-angle-spinning (HRMAS) 1H NMR spectroscopy was used to study metabolite changes in the CCM chemical shift region in rat glioma ex vivo during apoptosis induced by thymidine kinase-ganciclovir gene therapy. Cell density and apoptotic activity in the tumours were quantified by histological methods. HRMAS 1H NMR was able to resolve peaks from choline (Cho), glycerophosphocholine (GPC), phosphocholine (PC), taurine (Tau) and myo-inositol (myo-Ins), all of which contribute to the in vivo 1H NMR peak centred at 3.23 ppm. The early phase of apoptosis (treatment day 4), with a approximately 2.8-fold increase in the number of apoptotic nuclei (at constant cell density of 1.8 +/- 0.1 x 10(5) cells/mm3) was associated with increases in resonance intensity from GPC and PC, while Cho and Tau remained unchanged. Later stage apoptosis, accompanied by synchronous cell death (cell density declined to 0.7 +/- 0.02 x 10(5) cells/mm3), resulted in a significant decline in Tau relative to untreated tumours, while the contents of CCMs and myo-Ins detectable by 1H HRMAS were unchanged. These observations demonstrate that, while the in vivo 1H NMR peak at 3.23 ppm is indicative of cellular processes involved in apoptosis, the biochemical changes monitored by this resonance involve a number of different and chemically distinct metabolites.
Authors:
Piia K Valonen; Julian L Griffin; Kimmo K Lehtimäki; Timo Liimatainen; Jeremy K Nicholson; Olli H J Gröhn; Risto A Kauppinen
Related Documents :
20398626 - Potent antitumor activities of recombinant human pdcd5 protein in combination with chem...
9261566 - The apoptosis of hel cells induced by hydroxyurea.
15691586 - Apoptotic volume decrease and nitric oxide.
17606986 - Polarity proteins par6 and apkc regulate cell death through gsk-3beta in 3d epithelial ...
22804576 - Mechanisms of intracellular scaling.
14534786 - The cellular localization of prosystemin: a functional role for phloem parenchyma in sy...
Publication Detail:
Type:  Evaluation Studies; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  NMR in biomedicine     Volume:  18     ISSN:  0952-3480     ISO Abbreviation:  NMR Biomed     Publication Date:  2005 Jun 
Date Detail:
Created Date:  2005-06-30     Completed Date:  2005-09-16     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  8915233     Medline TA:  NMR Biomed     Country:  England    
Other Details:
Languages:  eng     Pagination:  252-9     Citation Subset:  IM    
Copyright Information:
Copyright 2005 John Wiley & Sons, Ltd
Affiliation:
Department of Biomedical NMR and National Bio-NMR Facility, A.I. Virtanen Institute for Molecular Sciences, University of Kuopio, Kuopio, Finland.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis*
Biological Markers / analysis,  metabolism
Brain Neoplasms / diagnosis,  genetics,  metabolism*,  therapy
Choline / analysis*
Female
Gene Therapy / methods*
Glioma / diagnosis,  genetics,  metabolism*,  therapy
Image Interpretation, Computer-Assisted / methods
Magnetic Resonance Imaging / methods*
Magnetic Resonance Spectroscopy / methods*
Protons
Rats
Spin Labels
Treatment Outcome
Chemical
Reg. No./Substance:
0/Biological Markers; 0/Protons; 0/Spin Labels; 62-49-7/Choline

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Mediotemporal contributions to semantic processing: fMRI evidence from ambiguity processing during s...
Next Document:  Perceptions of insurance coverage for screening mammography among women in need of screening.