Document Detail


Hepatic steatosis, low-grade chronic inflammation and hormone/growth factor/adipokine imbalance.
MedLine Citation:
PMID:  20939105     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Non-alcoholic fatty liver disease (NAFLD), a further expression of metabolic syndrome, strictly linked to obesity and diabetes mellitus, is characterized by insulin resistance (IR), elevated serum levels of free fatty acids and fatty infiltration of the liver, which is known as hepatic steatosis. Hepatocyte apoptosis is a key feature of this disease and correlates with its severity. Free-fatty-acid-induced toxicity represents one of mechanisms for the pathogenesis of NAFLD and hormones, growth factors and adipokines influence also play a key role. This review highlights the various pathways that contribute to the development of hepatic steatosis. Circulating concentrations of inflammatory cytokines are reckoned to be the most important factor in causing and maintaining IR. Low-grade chronic inflammation is fundamental in the progression of NAFLD toward higher risk cirrhotic states.
Authors:
Giovanni Tarantino; Silvia Savastano; Annamaria Colao
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  World journal of gastroenterology : WJG     Volume:  16     ISSN:  1007-9327     ISO Abbreviation:  World J. Gastroenterol.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-10-12     Completed Date:  2011-01-28     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100883448     Medline TA:  World J Gastroenterol     Country:  China    
Other Details:
Languages:  eng     Pagination:  4773-83     Citation Subset:  IM    
Affiliation:
Department of Clinical and Experimental Medicine, Federico II University Medical School of Naples, Italy. tarantin@unina.it
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MeSH Terms
Descriptor/Qualifier:
Adipokines / metabolism*
Animals
Cardiovascular Diseases / physiopathology
Chronic Disease
Fatty Liver / etiology,  physiopathology*
Hormones / metabolism*
Humans
Inflammation / metabolism*
Insulin Resistance / physiology
Intercellular Signaling Peptides and Proteins / metabolism*
Mitochondria / metabolism
Obesity / complications,  physiopathology
Risk Factors
Chemical
Reg. No./Substance:
0/Adipokines; 0/Hormones; 0/Intercellular Signaling Peptides and Proteins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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