Document Detail


Hepatic overexpression of cholesteryl ester hydrolase enhances cholesterol elimination and in vivo reverse cholesterol transport.
MedLine Citation:
PMID:  18599737     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Neutral cholesteryl ester hydrolase (CEH)-mediated hydrolysis of cellular cholesteryl esters (CEs) is required not only to generate free cholesterol (FC) for efflux from macrophages but also to release FC from lipoprotein-delivered CE in the liver for bile acid synthesis or direct secretion into the bile. We hypothesized that hepatic expression of CEH would regulate the hydrolysis of lipoprotein-derived CE and enhance reverse cholesterol transport (RCT). Adenoviral-mediated CEH overexpression led to a significant increase in bile acid output. To assess the role of hepatic CEH in promoting flux of cholesterol from macrophages to feces, cholesterol-loaded and [3H]cholesterol-labeled J774 macrophages were injected intraperitoneally into mice and the appearance of [3H]cholesterol in gallbladder bile and feces over 48 h was quantified. Mice overexpressing CEH had significantly higher [3H]cholesterol radiolabel in bile and feces, and it was associated with bile acids. This CEH-mediated increased movement of [3H]cholesterol from macrophages to bile acids and feces was significantly attenuated in SR-BI(-/-) mice. These studies demonstrate that similar to macrophage CEH that rate-limits the first step, hepatic CEH regulates the last step of RCT by promoting the flux of cholesterol entering the liver via SR-BI and increasing hepatic bile acid output.
Authors:
Bin Zhao; Jingmei Song; Shobha Ghosh
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-07-03
Journal Detail:
Title:  Journal of lipid research     Volume:  49     ISSN:  0022-2275     ISO Abbreviation:  J. Lipid Res.     Publication Date:  2008 Oct 
Date Detail:
Created Date:  2008-09-15     Completed Date:  2008-10-22     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0376606     Medline TA:  J Lipid Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2212-7     Citation Subset:  IM    
Affiliation:
Department of Internal Medicine, Virginia Commonwealth University Medical Center, Richmond, VA 23298-0050, USA.
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MeSH Terms
Descriptor/Qualifier:
Adenoviridae / genetics
Animals
Bile / metabolism
Bile Acids and Salts / metabolism
Biological Transport
Cell Line
Cholesterol / metabolism*
Cholesterol, HDL / metabolism
Feces
Gene Expression
Gene Expression Regulation, Enzymologic
Humans
Lipoproteins / blood
Liver / enzymology,  metabolism*
Macrophages / metabolism
Mice
Sterol Esterase / genetics,  metabolism*
Chemical
Reg. No./Substance:
0/Bile Acids and Salts; 0/Cholesterol, HDL; 0/Lipoproteins; 57-88-5/Cholesterol; EC 3.1.1.13/Sterol Esterase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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