Document Detail


Hep3B human hepatoma cells support replication of the wild-type and a 5'-end deletion mutant GB virus B replicon.
MedLine Citation:
PMID:  14581524     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hepatitis C virus (HCV) and GB virus B (GBV-B) replicons have been reported to replicate only in Huh7 cells. Here we demonstrate that subpopulations of another human hepatoma cell line, Hep3B, are permissive for the GBV-B replicon, showing different levels of enhancement of replication from those of the unselected parental cell population. Adaptive mutations are not required for replication of the GBV-B replicon in these cells, as already demonstrated for Huh7 cells. Nonetheless, we identified a mutant replicon in one of the selected cell lines, which, although lacking the 5' end proximal stem-loop, is able to replicate in Hep3B cells as well as in Huh7 cells. This mutant indeed shows a higher replication efficiency than does wild-type replicon, especially in the Hep3B cell clone from which it was originally recovered. This indicates that the stem-loop Ia is not necessary for replication of the GBV-B replicon in human cells, unlike what occurs with HCV, and that its absence can even provide a selective advantage.
Authors:
Amedeo De Tomassi; Maura Pizzuti; Cinzia Traboni
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Journal of virology     Volume:  77     ISSN:  0022-538X     ISO Abbreviation:  J. Virol.     Publication Date:  2003 Nov 
Date Detail:
Created Date:  2003-10-28     Completed Date:  2003-11-25     Revised Date:  2013-04-18    
Medline Journal Info:
Nlm Unique ID:  0113724     Medline TA:  J Virol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  11875-81     Citation Subset:  IM    
Affiliation:
Istituto di Ricerche di Biologia Molecolare P. Angeletti, 00040 Pomezia (Rome), Italy.
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MeSH Terms
Descriptor/Qualifier:
5' Untranslated Regions / chemistry
Carcinoma, Hepatocellular / virology*
Cell Line, Tumor
GB virus B / genetics,  physiology*
Humans
Liver Neoplasms / virology*
Mutation
Protein Biosynthesis
RNA, Viral / chemistry
Replicon*
Virus Replication*
Chemical
Reg. No./Substance:
0/5' Untranslated Regions; 0/RNA, Viral
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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