Document Detail

Heart failure in septic shock: effects of inotropic support.
MedLine Citation:
PMID:  2403514     Owner:  NLM     Status:  MEDLINE    
Many animal studies have attempted to simulate the circulatory responses to Gram-negative septicemia (iv infusion of live bacteria, fecal inoculation into body cavities, and administration of purified endotoxins by various routes), but the contribution of the heart to the adverse hemodynamic derangements and thus to the pathogenesis of shock is difficult to determine because of peripheral vascular events that influence cardiac performance. When blood pools in the periphery, venous return decreases and cardiac output can decrease without a primary myocardial defect being present. However, early heart dysfunction has been recognized in sepsis. Hemodynamic monitoring has not reduced overall mortality, but it has been helpful in guiding fluid administration and evaluating response to vasopressor therapy.
B Schremmer; J F Dhainaut
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Critical care medicine     Volume:  18     ISSN:  0090-3493     ISO Abbreviation:  Crit. Care Med.     Publication Date:  1990 Jan 
Date Detail:
Created Date:  1990-02-06     Completed Date:  1990-02-06     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0355501     Medline TA:  Crit Care Med     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  S49-55     Citation Subset:  AIM; IM    
Service de Réanimation Polyvalente, Cochin Port-Royal University Hospital, Paris, France.
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MeSH Terms
Cardiac Output
Combined Modality Therapy
Heart / radionuclide imaging
Heart Failure / etiology,  physiopathology*
Pulmonary Wedge Pressure
Shock, Septic / complications,  physiopathology*,  therapy
Stroke Volume

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