Document Detail


Heart failure and cachexia: insights offered from molecular biology.
MedLine Citation:
PMID:  17981550     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Chronic heart failure (CHF) is an enormous medical and communal burden. The syndrome is common, carries a grim prognosis and severely impacts quality of life. Those patients who develop cardiac cachexia combat both important disability and a poor outlook. Muscle wasting is a critical component of cachexia. The pathophysiological determinants are numerous and some of them are common to other chronic severe illnesses. There is increasing awareness, however, that heart failure related myopathy is a distinct entity, characterized by specific functional, structural and morphologic changes and the involvement of several neurohormonal pathways, catabolic processes, a pro-inflammatory environment and increased oxidative stress. Although clear-cut evidence based solutions for the problem are not readily available, the modulating effects of regular exercise in CHF patients suggest that physical training should at least be incorporated in the essentially multi-disciplinary approach.
Authors:
Viviane M A Conraads; Vicky Y Hoymans; Christiaan J Vrints
Publication Detail:
Type:  Journal Article; Review     Date:  2008-01-01
Journal Detail:
Title:  Frontiers in bioscience : a journal and virtual library     Volume:  13     ISSN:  1093-4715     ISO Abbreviation:  Front. Biosci.     Publication Date:  2008  
Date Detail:
Created Date:  2007-11-05     Completed Date:  2007-12-11     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9709506     Medline TA:  Front Biosci     Country:  United States    
Other Details:
Languages:  eng     Pagination:  325-35     Citation Subset:  IM    
Affiliation:
Department of Cardiology, Centre for Cell Therapy and Regenerative Medicine, University Hospital Antwerp, Belgium. Viviane.Conraads@uza.be
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MeSH Terms
Descriptor/Qualifier:
Animals
Cachexia / complications*
Catecholamines / metabolism
Exercise
Ghrelin / metabolism
Heart Failure / complications*
Humans
Inflammation
Leptin / metabolism
Muscle, Skeletal / metabolism
Neurosecretory Systems
Oxidative Stress
Physical Conditioning, Animal
Chemical
Reg. No./Substance:
0/Catecholamines; 0/Ghrelin; 0/Leptin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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