Document Detail


Histone deacetylase 6 and heat shock protein 90 control the functions of Foxp3(+) T-regulatory cells.
MedLine Citation:
PMID:  21444725     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Foxp3(+) T-regulatory cells (Tregs) are key to immune homeostasis such that their diminished numbers or function can cause autoimmunity and allograft rejection. Foxp3(+) Tregs express multiple histone/protein deacetylases (HDACs) that regulate chromatin remodeling, gene expression, and protein function. Pan-HDAC inhibitors developed for oncologic applications enhance Treg production and Treg suppression function but have limited nononcologic utility given their broad actions and various side effects. We show, using HDAC6-deficient mice and wild-type (WT) mice treated with HDAC6-specific inhibitors, that HDAC6 inhibition promotes Treg suppressive activity in models of inflammation and autoimmunity, including multiple forms of experimental colitis and fully major histocompatibility complex (MHC)-incompatible cardiac allograft rejection. Many of the beneficial effects of HDAC6 targeting are also achieved by inhibition of the HDAC6-regulated protein heat shock protein 90 (HSP90). Hence, selective targeting of a single HDAC isoform, HDAC6, or its downstream target, HSP90, can promote Treg-dependent suppression of autoimmunity and transplant rejection.
Authors:
Edwin F de Zoeten; Liqing Wang; Kyle Butler; Ulf H Beier; Tatiana Akimova; Hong Sai; James E Bradner; Ralph Mazitschek; Alan P Kozikowski; Patrick Matthias; Wayne W Hancock
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2011-03-28
Journal Detail:
Title:  Molecular and cellular biology     Volume:  31     ISSN:  1098-5549     ISO Abbreviation:  Mol. Cell. Biol.     Publication Date:  2011 May 
Date Detail:
Created Date:  2011-04-28     Completed Date:  2011-06-27     Revised Date:  2012-04-16    
Medline Journal Info:
Nlm Unique ID:  8109087     Medline TA:  Mol Cell Biol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2066-78     Citation Subset:  IM    
Affiliation:
Division of Gastroenterology, Hepatology and Nutrition, Department of Pediatrics, Children's Hospital of Philadelphia and University of Pennsylvania School of Medicine, 3615 Civic Center Boulevard, Philadelphia, Pennsylvania 19104-4318, USA.
Data Bank Information
Bank Name/Acc. No.:
GEO/GSE27896
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MeSH Terms
Descriptor/Qualifier:
Anilides / pharmacology
Animals
Autoimmunity
Blotting, Western
Colitis / metabolism
Disease Progression
Flow Cytometry
Forkhead Transcription Factors / analysis
Gene Targeting
Graft Rejection / prevention & control
HSP90 Heat-Shock Proteins / antagonists & inhibitors,  genetics,  metabolism*
Histone Deacetylase Inhibitors / pharmacology
Histone Deacetylases / genetics,  metabolism*
Hydroxamic Acids / pharmacology
Immunoprecipitation
Indoles / pharmacology
Mice
Mice, Inbred BALB C
Mice, Inbred C57BL
Mice, Knockout
Microarray Analysis
Molecular Sequence Data
Polymerase Chain Reaction
T-Lymphocytes, Regulatory / drug effects,  immunology*,  metabolism
Transplantation Tolerance
Grant Support
ID/Acronym/Agency:
K08 CA128972-04/CA/NCI NIH HHS; K08 CA128972-05/CA/NCI NIH HHS; K08DK080189/DK/NIDDK NIH HHS; P01AI073489/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Anilides; 0/Forkhead Transcription Factors; 0/Foxp3 protein, mouse; 0/HSP90 Heat-Shock Proteins; 0/Histone Deacetylase Inhibitors; 0/Hydroxamic Acids; 0/Indoles; 0/tubacin; 0/tubastatin A; EC 3.5.1.98/Hdac6 protein, mouse; EC 3.5.1.98/Histone Deacetylases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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