Document Detail


Gut adaptation and the insulin-like growth factor system: regulation by glutamine and IGF-I administration.
MedLine Citation:
PMID:  8944702     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Intestinal adaptation after extensive small bowel resection in rats is augmented by the provision of diets supplemented with the amino acid glutamine (Gln) or by administration of insulin-like growth factor-I (IGF-I). The goal of this study was to investigate potential synergistic effects of Gln and IGF-I on postresection ileal hyperplasia. Rats underwent 80% small bowel resection (SBR) and then were fed low-Gln or L-Gln-enriched diets and subcutaneously given recombinant human IGF-I or vehicle for 7 days. Gln and IGF-I each significantly enhanced adaptive ileal hyperplasia (DNA content) compared with rats receiving vehicle and low-Gln diet. Ileal DNA content was highest when IGF-I was administered together with Gln supplementation. Combined IGF-I plus Gln synergistically increased ileal weight and protein content. This was associated with higher plasma concentrations of IGF-I and Gln than observed when IGF-I or Gln was given individually. Ileal IGF-I mRNA expression rose nearly twofold during gut adaptation after SBR; this response was augmented with IGF-I administration but was unaltered by Gln feeding. In contrast, dietary Gln, but not IGF-I therapy, prevented a decrease in hepatic IGF-I mRNA induced by SBR. We conclude that parenteral IGF-I and enteral Gln have both individual and synergistic effects on ileal adaptation after massive small intestinal resection. These findings support the concept that specific gut-trophic nutrients and growth factors may be combined to enhance intestinal adaptation and possibly reduce the severity of short bowel syndrome after intestinal resection.
Authors:
T R Ziegler; M P Mantell; J C Chow; J L Rombeau; R J Smith
Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  The American journal of physiology     Volume:  271     ISSN:  0002-9513     ISO Abbreviation:  Am. J. Physiol.     Publication Date:  1996 Nov 
Date Detail:
Created Date:  1997-01-09     Completed Date:  1997-01-09     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0370511     Medline TA:  Am J Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  G866-75     Citation Subset:  IM    
Affiliation:
Joslin Research Laboratory, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts 02215, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Enteral Nutrition
Food, Fortified*
Glutamine / pharmacology*
Humans
Hyperplasia
Ileum / drug effects,  pathology,  physiology*
Insulin-Like Growth Factor Binding Protein 3 / biosynthesis
Insulin-Like Growth Factor Binding Protein 4 / biosynthesis
Insulin-Like Growth Factor I / biosynthesis*,  metabolism,  pharmacology*
Intestine, Small / physiology*,  surgery
Liver / drug effects,  physiology
Male
Muscle, Smooth / drug effects,  pathology,  physiology
RNA, Messenger / biosynthesis
Rats
Rats, Sprague-Dawley
Recombinant Proteins / pharmacology
Transcription, Genetic* / drug effects
Grant Support
ID/Acronym/Agency:
DK-43038/DK/NIDDK NIH HHS; DK-48503/DK/NIDDK NIH HHS; GM-36428/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/Insulin-Like Growth Factor Binding Protein 3; 0/Insulin-Like Growth Factor Binding Protein 4; 0/RNA, Messenger; 0/Recombinant Proteins; 56-85-9/Glutamine; 67763-96-6/Insulin-Like Growth Factor I

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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