Document Detail


Glyceroneogenesis and the supply of glycerol-3-phosphate for glyceride-glycerol synthesis in liver slices of fasted and diabetic rats.
MedLine Citation:
PMID:  17726141     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The pathways of glycerol-3-phosphate (G3P) generation for glyceride synthesis were examined in precision-cut liver slices of fasted and diabetic rats. The incorporation of 5 mM [U-(14)C]glucose into glyceride-glycerol, used to evaluate G3P generation via glycolysis, was reduced by approximately 26-36% in liver slices of fasted and diabetic rats. The glycolytic flux was reduced by approximately 60% in both groups. The incorporation of 1.0 mM [2-(14)C]pyruvate into glyceride-glycerol (glyceroneogenesis) increased approximately 50% and approximately 36% in slices of fasted and diabetic rats, respectively, which also showed a two-fold increase in the activity phosphoenolpyruvate carboxykinase. The increased incorporation of 1.0 mM [2-(14)C]pyruvate into glyceride-glycerol by slices of fasted rats was not affected by the addition of 5 mM glucose to the incubation medium. The activity of glycerokinase and the incorporation of 1 mM [U-(14)C]glycerol into glyceride-glycerol, evaluators of G3P formation by direct glycerol phosphorylation, did not differ significantly from controls in slices of the two experimental groups. Rates of incorporation of 1 mM [2-(14)C]pyruvate and [U-(14)C]glycerol into glucose of incubation medium (gluconeogenesis) were approximately 140 and approximately 20% higher in fasted and diabetic slices than in control slices. It could be estimated that glyceroneogenesis by liver slices of fasted rats contributed with approximately 20% of G3P generated for glyceride-glycerol synthesis, the glycolytic pathway with approximately 5%, and direct phosphorylation of glycerol by glycerokinase with approximately 75%. Pyruvate contributed with 54% and glycerol with 46% of gluconeogenesis. The present data indicate that glyceroneogenesis has a significant participation in the generation of G3P needed for the increased glyceride-glycerol synthesis in liver during fasting and diabetes.
Authors:
Maria Emilia Soares Martins-Santos; Valéria Ernestânia Chaves; Danúbia Frasson; Renata Polessi Boschini; Maria Antonieta Rissato Garófalo; Isis do Carmo Kettelhut; Renato Hélios Migliorini
Publication Detail:
Type:  In Vitro; Journal Article; Research Support, Non-U.S. Gov't     Date:  2007-08-28
Journal Detail:
Title:  American journal of physiology. Endocrinology and metabolism     Volume:  293     ISSN:  0193-1849     ISO Abbreviation:  Am. J. Physiol. Endocrinol. Metab.     Publication Date:  2007 Nov 
Date Detail:
Created Date:  2007-11-08     Completed Date:  2007-12-18     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100901226     Medline TA:  Am J Physiol Endocrinol Metab     Country:  United States    
Other Details:
Languages:  eng     Pagination:  E1352-7     Citation Subset:  IM    
Affiliation:
Department of Biochemistry and Immunology, School of Medicine, USP 14049-900 Ribeirão Preto, São Paulo, Brazil.
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MeSH Terms
Descriptor/Qualifier:
Adenosine Triphosphate / metabolism
Animals
Carbon Radioisotopes / diagnostic use
Diabetes Mellitus, Experimental / enzymology,  metabolism*
Food Deprivation / physiology
Glucose / metabolism
Glycerides / biosynthesis*
Glycerol / metabolism
Glycerol Kinase / metabolism
Glycerophosphates / metabolism*
Liver / enzymology,  metabolism*
Male
Phosphoenolpyruvate Carboxykinase (ATP) / metabolism
Pyruvic Acid / metabolism
Rats
Rats, Wistar
Triglycerides / metabolism
Chemical
Reg. No./Substance:
0/Carbon Radioisotopes; 0/Glycerides; 0/Glycerophosphates; 0/Triglycerides; 127-17-3/Pyruvic Acid; 50-99-7/Glucose; 56-65-5/Adenosine Triphosphate; 56-81-5/Glycerol; 57-03-4/alpha-glycerophosphoric acid; EC 2.7.1.30/Glycerol Kinase; EC 4.1.1.49/Phosphoenolpyruvate Carboxykinase (ATP)

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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