Document Detail


Ghrelin treatment protects lactotrophs from apoptosis in the pituitary of diabetic rats.
MedLine Citation:
PMID:  19540304     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Poorly controlled diabetes is associated with hormonal imbalances, including decreased prolactin production partially due to increased lactotroph apoptosis. In addition to its metabolic actions, ghrelin inhibits apoptosis in several cell types. Thus, we analyzed ghrelin's effects on diabetes-induced pituitary cell death and hormonal changes. Six weeks after onset of diabetes in male Wistar rats (streptozotocin 70 mg/kg), minipumps infusing saline or 24 nmol ghrelin/day were implanted (jugular). Rats were killed two weeks later. Ghrelin did not modify body weight or serum glucose, leptin or adiponectin, but increased total ghrelin (P<0.05), IGF-I (P<0.01) and prolactin (P<0.01) levels. Ghrelin decreased cell death, iNOS and active caspase-8 (P<0.05) and increased prolactin (P<0.05), Bcl-2 (P<0.01) and Hsp70 (P<0.05) content in the pituitary. In conclusion, ghrelin prevents diabetes-induced death of lactotrophs, decreasing caspase-8 activation and iNOS content and increasing anti-apoptotic pathways such as pituitary Bcl-2 and Hsp70 and serum IGF-I concentrations.
Authors:
M Granado; J A Chowen; C García-Cáceres; A Delgado-Rubín; V Barrios; E Castillero; J Argente; L M Frago
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-06-18
Journal Detail:
Title:  Molecular and cellular endocrinology     Volume:  309     ISSN:  1872-8057     ISO Abbreviation:  Mol. Cell. Endocrinol.     Publication Date:  2009 Oct 
Date Detail:
Created Date:  2009-08-03     Completed Date:  2009-10-26     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7500844     Medline TA:  Mol Cell Endocrinol     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  67-75     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, Universidad Autónoma de Madrid, Madrid, Spain.
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MeSH Terms
Descriptor/Qualifier:
Adiponectin / blood
Animals
Apoptosis / drug effects*
Biological Markers / metabolism
Body Weight / drug effects
Caspases / metabolism
Cytoprotection / drug effects*
Diabetes Mellitus, Experimental / blood,  pathology*
Ghrelin / blood,  pharmacology*
HSP70 Heat-Shock Proteins / metabolism
In Situ Nick-End Labeling
Insulin / blood
Insulin-Like Growth Factor I / metabolism
Lactotrophs / cytology*,  drug effects*,  enzymology
Leptin / blood
Nitric Oxide Synthase Type II / metabolism
Prolactin / metabolism
Proto-Oncogene Proteins c-bcl-2 / metabolism
Rats
Weight Gain / drug effects
X-Linked Inhibitor of Apoptosis Protein / metabolism
Chemical
Reg. No./Substance:
0/Adiponectin; 0/Biological Markers; 0/Ghrelin; 0/HSP70 Heat-Shock Proteins; 0/Leptin; 0/Proto-Oncogene Proteins c-bcl-2; 0/X-Linked Inhibitor of Apoptosis Protein; 11061-68-0/Insulin; 67763-96-6/Insulin-Like Growth Factor I; 9002-62-4/Prolactin; EC 1.14.13.39/Nitric Oxide Synthase Type II; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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