Document Detail


Getting back at nature: understanding thymic development and overcoming its atrophy.
MedLine Citation:
PMID:  20483662     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
T cell development is a complex and tightly regulated process involving reciprocal interactions between the thymic stroma and differentiating thymocytes. Normal thymic function is critical for immunity and microenvironmental defects predispose to dysregulation in the T cell compartment. Thymic structure and function are also severely damaged by chemotherapy and pre-transplant conditioning. Furthermore, poor immune competence with ageing is closely linked to thymic atrophy. Overcoming such thymic defects would have immediate application in many diseases, especially the recovery of cancer patients from cytotoxic treatment. Reversing the thymus ageing process via inhibition of atrophic factors such as sex steroids or administration of thymopoietic growth factors is one possible approach. Moreover, it is becoming clear a common thymic epithelial progenitor exists, raising the possibility for de novo thymus generation using emerging stem cell and tissue engineering technologies. Achievement of this goal will open up many avenues for the application of thymus-based immune rejuvenation and manipulation.
Authors:
Tracy S P Heng; Ann P Chidgey; Richard L Boyd
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2010-05-17
Journal Detail:
Title:  Current opinion in pharmacology     Volume:  10     ISSN:  1471-4973     ISO Abbreviation:  Curr Opin Pharmacol     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-08-03     Completed Date:  2010-11-12     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100966133     Medline TA:  Curr Opin Pharmacol     Country:  England    
Other Details:
Languages:  eng     Pagination:  425-33     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Ltd. All rights reserved.
Affiliation:
Monash Immunology and Stem Cell Laboratories, Monash University, Clayton, VIC 3800, Australia.
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MeSH Terms
Descriptor/Qualifier:
Aging / immunology,  physiology*
Animals
Antineoplastic Agents / toxicity
Atrophy / chemically induced
Epithelial Cells
Fibroblast Growth Factor 7 / pharmacology
Gonadal Steroid Hormones / antagonists & inhibitors
Growth Hormone / pharmacology
Humans
Insulin-Like Growth Factor I / pharmacology
Interleukin-7 / pharmacology
Membrane Proteins / pharmacology
Stem Cells / physiology
Stromal Cells
T-Lymphocytes / drug effects,  immunology,  physiology*
Thymus Gland / drug effects,  immunology,  physiology*
fms-Like Tyrosine Kinase 3 / metabolism
Chemical
Reg. No./Substance:
0/Antineoplastic Agents; 0/Gonadal Steroid Hormones; 0/Interleukin-7; 0/Membrane Proteins; 0/flt3 ligand protein; 126469-10-1/Fibroblast Growth Factor 7; 67763-96-6/Insulin-Like Growth Factor I; 9002-72-6/Growth Hormone; EC 2.7.10.1/FLT3 protein, human; EC 2.7.10.1/fms-Like Tyrosine Kinase 3

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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