Document Detail


Gestational glucose tolerance and cord blood leptin levels predict slower weight gain in early infancy.
MedLine Citation:
PMID:  20855080     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: To determine the extent to which known prenatal and perinatal predictors of childhood obesity also predict weight gain in early infancy.
STUDY DESIGN: We studied 690 infants participating in the prospective cohort Project Viva. We measured length and weight at birth and at 6 months. Using multivariable linear regression, we examined relationships of selected maternal and infant factors with change in weight-for-length z-score (WFL-z) from 0 to 6 months.
RESULTS: Mean (standard deviation) change in WFL-z from 0 to 6 months was 0.23 (1.11), which translates to 4500 grams gained from birth to 6 months of life in an infant with average birth weight and length. After adjustment for confounding variables and birth weight-for-gestational age z-score (-0.28 [95% confidence interval, -0.37, -0.19] per unit), cord blood leptin (-0.40 [95%confidence interval, -0.61, -0.19] per 10 ng/mL), and gestational diabetes -0.50 [95%confidence interval, -0.88, -0.11] versus normal glucose tolerance)were each associated with slower gain in WFL-z from 0 to 6 months.
CONCLUSIONS: Higher neonatal leptin and gestational diabetes predicted slower weight gain in the first 6 months of life. The hormonal milieu of the intrauterine environment may determine growth patterns in early infancy and thus later obesity.
Authors:
Margaret Parker; Sheryl L Rifas-Shiman; Mandy B Belfort; Elsie M Taveras; Emily Oken; Christos Mantzoros; Matthew W Gillman
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-09-19
Journal Detail:
Title:  The Journal of pediatrics     Volume:  158     ISSN:  1097-6833     ISO Abbreviation:  J. Pediatr.     Publication Date:  2011 Feb 
Date Detail:
Created Date:  2011-01-17     Completed Date:  2011-02-24     Revised Date:  2011-10-19    
Medline Journal Info:
Nlm Unique ID:  0375410     Medline TA:  J Pediatr     Country:  United States    
Other Details:
Languages:  eng     Pagination:  227-33     Citation Subset:  AIM; IM    
Affiliation:
Division of Newborn Medicine, Children's Hospital Boston, Harvard Medical School, Boston, MA, USA. margaret.parker@childrens.harvard.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Biological Markers / blood
Blood Glucose / analysis*
Body Height
Body Mass Index
Child Development / physiology
Cohort Studies
Confidence Intervals
Diabetes, Gestational / blood*,  physiopathology
Female
Fetal Blood / chemistry*
Follow-Up Studies
Gestational Age
Glucose Intolerance
Glucose Tolerance Test
Humans
Infant
Infant, Newborn
Leptin / blood*
Male
Multivariate Analysis
Odds Ratio
Predictive Value of Tests
Pregnancy
Prospective Studies
Regression Analysis
Sensitivity and Specificity
Time Factors
Weight Gain / physiology*
Grant Support
ID/Acronym/Agency:
HD034568/HD/NICHD NIH HHS; HL 064925/HL/NHLBI NIH HHS; HL 068041/HL/NHLBI NIH HHS; P30 DK040561-15/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Biological Markers; 0/Blood Glucose; 0/Leptin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Leukocyte telomere length in healthy Caucasian and African-American adolescents: relationships with ...
Next Document:  Liver Metabolomic Changes Identify Biochemical Pathways in Hemorrhagic Shock.