Document Detail


Genomic imprinting of experience-dependent cortical plasticity by the ubiquitin ligase gene Ube3a.
MedLine Citation:
PMID:  20212164     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
A defect in the maternal copy of a ubiqutin ligase gene Ube3a can produce a neurodevelopmental defect in human children known as Angelman syndrome. We investigated the role of the maternally expressed Ube3a gene in experience-dependent development and plasticity of the mouse visual system. As demonstrated by optical imaging, rapid ocular dominance (OD) plasticity after brief monocular deprivation (MD) was severely impaired during the critical period (CP) in the visual cortex (VC) of Ube3a maternal-deficient (m-/p+) mice. Prolonged MD elicited significant plasticity in m-/p+ mice that never matched the level seen in control animals. In older animals after the CP, 7-day MD elicited mild OD shifts in both control and m-/p+ mice; however, the OD shifts in m-/p+ mice lacked the strengthening of visual responses to the two eyes characteristic of normal adult plasticity. Anatomic effects of the maternal deficiency include reduced spine density on basal, but not apical, dendrites of pyramidal neurons in the binocular region of the VC. Imprinting of Ube3a expression was not fully established in the early postnatal period, consistent with the normal development of cortical retinotopy and visual acuity that we observed in m-/p+ mice, but was fully established by the onset of the CP. These results demonstrate that paternal and maternal genomes are not functionally equivalent for cortical plasticity, and that maternally expressed Ube3a is required for normal experience-dependent modification of cortical circuits during and after the CP.
Authors:
Masaaki Sato; Michael P Stryker
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-03-08
Journal Detail:
Title:  Proceedings of the National Academy of Sciences of the United States of America     Volume:  107     ISSN:  1091-6490     ISO Abbreviation:  Proc. Natl. Acad. Sci. U.S.A.     Publication Date:  2010 Mar 
Date Detail:
Created Date:  2010-03-24     Completed Date:  2010-04-16     Revised Date:  2010-09-24    
Medline Journal Info:
Nlm Unique ID:  7505876     Medline TA:  Proc Natl Acad Sci U S A     Country:  United States    
Other Details:
Languages:  eng     Pagination:  5611-6     Citation Subset:  IM    
Affiliation:
W.M. Keck Foundation Center for Integrative Neuroscience, Department of Physiology, University of California, San Francisco, CA 94143-0444, USA.
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MeSH Terms
Descriptor/Qualifier:
Angelman Syndrome / genetics,  physiopathology
Animals
Dominance, Ocular / genetics,  physiology
Female
Genomic Imprinting*
Humans
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Neuronal Plasticity / genetics,  physiology
Ubiquitin-Protein Ligases / deficiency,  genetics*,  physiology
Visual Cortex / growth & development,  physiology*
Grant Support
ID/Acronym/Agency:
EY02874/EY/NEI NIH HHS; MH077972/MH/NIMH NIH HHS
Chemical
Reg. No./Substance:
EC 6.3.2.19/Ube3a protein, mouse; EC 6.3.2.19/Ubiquitin-Protein Ligases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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