Document Detail


Genetic analysis of congenital cystic adenomatoid malformation reveals a novel pulmonary gene: fatty acid binding protein-7 (brain type).
MedLine Citation:
PMID:  18391847     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The pathogenesis of congenital cystic adenomatoid malformation (CCAM) is unknown and its natural history is unpredictable. Fatty acid binding protein-7 (FABP-7) has been previously described in brain and breast development, but never before in the lung. We investigate gene expression in CCAM, and hypothesize that CCAM results from an aberration in the signaling pathway during lung development. Under IRB approval, tissue specimens of fetal CCAM, fetal control, postnatal CCAM, and postnatal control were examined and microarray analysis was performed. Candidate differentially expressed genes were selected with log-odds ratio (B) >0 and false discovery rate <0.05. Validation of differential expression was achieved at the RNA and protein levels. FABP-7 was underexpressed in fetal CCAM compared with fetal lung in both the microarray and by RT-PCR. Findings were duplicated by Western Blot analysis and immunohistochemistry. This is the first description of FABP-7 in the human lung. Decreased expression of FABP-7 in fetal CCAM compared with normal fetal lung at both the RNA and protein levels suggests FABP-7 may have a role in pulmonary development and in the pathogenesis of CCAM.
Authors:
Amy J Wagner; Amber Stumbaugh; Zachary Tigue; Jess Edmondson; Agnes C Paquet; Diana L Farmer; Samuel Hawgood
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Pediatric research     Volume:  64     ISSN:  0031-3998     ISO Abbreviation:  Pediatr. Res.     Publication Date:  2008 Jul 
Date Detail:
Created Date:  2008-06-26     Completed Date:  2008-08-21     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0100714     Medline TA:  Pediatr Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  11-6     Citation Subset:  IM    
Affiliation:
Department of Surgery, University of California, San Francisco, San Francisco, California 94143, USA.
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MeSH Terms
Descriptor/Qualifier:
Blotting, Western
Carrier Proteins / analysis,  genetics*
Child
Child, Preschool
Cystic Adenomatoid Malformation of Lung, Congenital / embryology,  genetics*,  metabolism
Down-Regulation
Female
Gene Expression Profiling / methods
Gene Expression Regulation, Developmental*
Genetic Predisposition to Disease
Gestational Age
Humans
Immunohistochemistry
Infant
Infant, Newborn
Lung / chemistry,  embryology
Male
Oligonucleotide Array Sequence Analysis
Reproducibility of Results
Reverse Transcriptase Polymerase Chain Reaction
Tumor Suppressor Proteins / analysis,  genetics*
Grant Support
ID/Acronym/Agency:
HL072301/HL/NHLBI NIH HHS; RR-00083/RR/NCRR NIH HHS
Chemical
Reg. No./Substance:
0/Carrier Proteins; 0/FABP7 protein, human; 0/Tumor Suppressor Proteins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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