Document Detail


Gastrointestinal permeability and absorptive capacity in sepsis.
MedLine Citation:
PMID:  8674326     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: To assess gastrointestinal permeability and functional absorptive capacity in patients with sepsis. DESIGN: Case control study to analyze gastrointestinal permeability and functional absorptive capacity of septic patients by differential saccharide absorption (from an oral test solution) and excretion. SETTING: The intensive Therapy Unit of St. Thomas' Hospital, London, UK. PATIENTS: Twenty patients with a mean Acute Physiology and Chronic Health Evaluation (APACHE) II score of 18.4 who were admitted to the intensive care unit with a diagnosis of sepsis. All patients were on enteral feeding. Patients with abdominal pathology were excluded. INTERVENTIONS: An oral test solution containing 5 g of lactulose, 1 g of L-rhamnose, 0.5 g of D-xylose, and 0.2 g of 3-O-methyl-D-glucose dissolved in water to a final volume of 100 mL was administered to patients and controls. Urine was collected for 5 hrs starting immediately after administration of the test solution and the saccharide content of the urine was estimated and expressed as a percentage recovery of the oral test solution. MEASUREMENTS AND MAIN RESULTS: Septic patients had increased lactulose/L-rhamnose urine excretion ratios (0.23 +/- 0.19) compared with control subjects (0.03 +/- 0.01, p < .001), consistent with increased gastrointestinal permeability in sepsis. Septic patients had decreased L-rhamnose/3-O-methyl-D-glucose urine excretion ratios (0.14 +/- 0.07) compared with normal controls (0.28 +/- 0.08, p < .001), consistent with decreased gastrointestinal functional absorptive capacity in sepsis. CONCLUSIONS: Patients with acute sepsis exhibit increased gastrointestinal permeability and decreased gastrointestinal functional absorptive capacity in comparison with healthy control subjects. These abnormalities may contribute to the pathophysiology of sepsis.
Authors:
J D Johnston; C J Harvey; I S Menzies; D F Treacher
Related Documents :
18074416 - Quality of life outcome of critical care survivors eighteen months after discharge from...
15962126 - Beyond sepsis pathophysiology with cytokines: what is their value as biomarkers for dis...
20439346 - Distinguishing myelodysplastic syndromes (mds) from idiopathic cytopenia of undetermine...
17699446 - North east italian prospective hospital renal outcome survey on acute kidney injury (ne...
22813886 - Patient cost associated with filling a prescription for extended-duration venous thromb...
23563936 - Is minimal residual disease monitoring clinically relevant in adults with acute myeloge...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Critical care medicine     Volume:  24     ISSN:  0090-3493     ISO Abbreviation:  Crit. Care Med.     Publication Date:  1996 Jul 
Date Detail:
Created Date:  1996-08-09     Completed Date:  1996-08-09     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0355501     Medline TA:  Crit Care Med     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1144-9     Citation Subset:  AIM; IM    
Affiliation:
Department of Chemical Pathology, United Medical School, St. Thomas' Hospital, UK.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
3-O-Methylglucose
Absorption
Acute Disease
Adult
Aged
Digestive System / physiopathology*
Female
Humans
Intestinal Absorption / physiology*
Lactulose / metabolism
Male
Methylglucosides / metabolism
Middle Aged
Permeability
Rhamnose / metabolism
Sepsis / physiopathology*
Xylose / metabolism
Chemical
Reg. No./Substance:
0/Methylglucosides; 0/Xylose; 10485-94-6/Rhamnose; 146-72-5/3-O-Methylglucose; 4618-18-2/Lactulose

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Inflammatory cytokine and nitric oxide responses in pediatric sepsis and organ failure.
Next Document:  Renal hemodynamics during norepinephrine and low-dose dopamine infusions in man.