Document Detail


A G1 checkpoint mediated by the retinoblastoma protein that is dispensable in terminal differentiation but essential for senescence.
MedLine Citation:
PMID:  20008551     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Terminally differentiated cell types are needed to live and function in a postmitotic state for a lifetime. Cellular senescence is another type of permanent arrest that blocks the proliferation of cells in response to genotoxic stress. Here we show that the retinoblastoma protein (pRB) uses a mechanism to block DNA replication in senescence that is distinct from its role in permanent cell cycle exit associated with terminal differentiation. Our work demonstrates that a subtle mutation in pRB that cripples its ability to interact with chromatin regulators impairs heterochromatinization and repression of E2F-responsive promoters during senescence. In contrast, terminally differentiated nerve and muscle cells bearing the same mutation fully exit the cell cycle and block E2F-responsive gene expression by a different mechanism. Remarkably, this reveals that pRB recruits chromatin regulators primarily to engage a stress-responsive G(1) arrest program.
Authors:
Srikanth Talluri; Christian E Isaac; Mohammad Ahmad; Shauna A Henley; Sarah M Francis; Alison L Martens; Rod Bremner; Frederick A Dick
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-12-14
Journal Detail:
Title:  Molecular and cellular biology     Volume:  30     ISSN:  1098-5549     ISO Abbreviation:  Mol. Cell. Biol.     Publication Date:  2010 Feb 
Date Detail:
Created Date:  2010-01-27     Completed Date:  2010-02-12     Revised Date:  2010-09-28    
Medline Journal Info:
Nlm Unique ID:  8109087     Medline TA:  Mol Cell Biol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  948-60     Citation Subset:  IM    
Affiliation:
Department of Biochemistry, University of Western Ontario, London, Ontario, Canada.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Aging*
Cell Differentiation*
Cell Line
DNA Replication
E2F Transcription Factors / genetics,  metabolism
Fibroblasts / cytology,  metabolism
G1 Phase*
Heterochromatin / metabolism
Humans
Mice
Mice, Knockout
Mutation
Promoter Regions, Genetic
Retinoblastoma Protein / deficiency,  genetics,  metabolism*
Transcription, Genetic
Grant Support
ID/Acronym/Agency:
MOP-64243//Canadian Institutes of Health Research; MOP-64253//Canadian Institutes of Health Research
Chemical
Reg. No./Substance:
0/E2F Transcription Factors; 0/Heterochromatin; 0/Retinoblastoma Protein
Comments/Corrections

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