Document Detail


G-CSF-mediated thrombopoietin release triggers neutrophil motility and mobilization from bone marrow via induction of Cxcr2 ligands.
MedLine Citation:
PMID:  21224471     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Emergency mobilization of neutrophil granulocytes (neutrophils) from the bone marrow (BM) is a key event of early cellular immunity. The hematopoietic cytokine granulocyte-colony stimulating factor (G-CSF) stimulates this process, but it is unknown how individual neutrophils respond in situ. We show by intravital 2-photon microscopy that a systemic dose of human clinical-grade G-CSF rapidly induces the motility and entry of neutrophils into blood vessels within the tibial BM of mice. Simultaneously, the neutrophil-attracting chemokine KC (Cxcl1) spikes in the blood. In mice lacking the KC receptor Cxcr2, G-CSF fails to mobilize neutrophils and antibody blockade of Cxcr2 inhibits the mobilization and induction of neutrophil motility in the BM. KC is expressed by megakaryocytes and endothelial cells in situ and is released in vitro by megakaryocytes isolated directly from BM. This production of KC is strongly increased by thrombopoietin (TPO). Systemic G-CSF rapidly induces the increased production of TPO in BM. Accordingly, a single injection of TPO mobilizes neutrophils with kinetics similar to G-CSF, and mice lacking the TPO receptor show impaired neutrophil mobilization after short-term G-CSF administration. Thus, a network of signaling molecules, chemokines, and cells controls neutrophil release from the BM, and their mobilization involves rapidly induced Cxcr2-mediated motility controlled by TPO as a pacemaker.
Authors:
Anja Köhler; Katia De Filippo; Mike Hasenberg; Cindy van den Brandt; Emma Nye; Martin P Hosking; Thomas E Lane; Linda Männ; Richard M Ransohoff; Anja E Hauser; Oliver Winter; Burkhart Schraven; Hartmut Geiger; Nancy Hogg; Matthias Gunzer
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2011-01-11
Journal Detail:
Title:  Blood     Volume:  117     ISSN:  1528-0020     ISO Abbreviation:  Blood     Publication Date:  2011 Apr 
Date Detail:
Created Date:  2011-04-22     Completed Date:  2011-07-13     Revised Date:  2012-04-23    
Medline Journal Info:
Nlm Unique ID:  7603509     Medline TA:  Blood     Country:  United States    
Other Details:
Languages:  eng     Pagination:  4349-57     Citation Subset:  AIM; IM    
Affiliation:
Institute of Molecular and Clinical Immunology, Otto-von-Guericke University, Magdeburg, Germany.
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MeSH Terms
Descriptor/Qualifier:
Animals
Bone Marrow / immunology
Bone Marrow Cells / cytology*,  immunology
Bone and Bones / cytology
Cell Line
Cell Movement
Cells, Cultured
Granulocyte Colony-Stimulating Factor / immunology*
Humans
Megakaryocytes / cytology,  immunology
Mice
Mice, Inbred C57BL
Neutrophils / cytology*,  immunology
Receptors, Interleukin-8B / immunology*
Thrombopoietin / immunology*
Grant Support
ID/Acronym/Agency:
DK077762/DK/NIDDK NIH HHS; HL076604/HL/NHLBI NIH HHS; //Cancer Research UK
Chemical
Reg. No./Substance:
0/Receptors, Interleukin-8B; 143011-72-7/Granulocyte Colony-Stimulating Factor; 9014-42-0/Thrombopoietin
Comments/Corrections
Comment In:
Blood. 2011 Apr 21;117(16):4166-7   [PMID:  21511963 ]

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