Document Detail


Functional interaction between TGF-beta and IL-12 in human primary allogeneic cytotoxicity and proliferative response.
MedLine Citation:
PMID:  8977184     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
IL-12 is an important cytokine in the control of cell-mediated immunity. We have investigated the functional interaction of IL-12 with TGF-beta1, a cytokine involved in the regulation of growth and differentiation of immunocompetent cells, during human allogeneic response development. Using primary MLR, our data show that addition of exogenous TGF-beta at the sensitizing phase of the primary MLR resulted in the inhibition of both allogeneic cytotoxic and proliferative responses. The inhibitory effect of TGF-beta on allogeneic response involves an abrogation of IL-12/p70 production upon allostimulation. In contrast to its effect on IL-12 production, TGF-beta did not alter the expression of IL-12R beta1-chain (IL-12R beta) in T cells induced upon allogeneic activation. Addition of exogenous IL-12 or IFN-gamma in the MLR cultures in the presence of TGF-beta did not result in reversal of CTL generation and T cell proliferation. Interestingly, TGF-beta was efficient in down-regulating IL-12 responsiveness of alloactivated T cells as well as TCR-alphabeta or TCR-gammadelta alloreactive T cell clones. These studies suggest that the inhibitory effect of TGF-beta on the development of human allogeneic proliferation and cytotoxic responses involves an additional mechanism associated with an interference with IL-12 pathway.
Authors:
C Pardoux; C Asselin-Paturel; J Chehimi; F Gay; F Mami-Chouaib; S Chouaib
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Journal of immunology (Baltimore, Md. : 1950)     Volume:  158     ISSN:  0022-1767     ISO Abbreviation:  J. Immunol.     Publication Date:  1997 Jan 
Date Detail:
Created Date:  1997-01-30     Completed Date:  1997-01-30     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  2985117R     Medline TA:  J Immunol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  136-43     Citation Subset:  AIM; IM    
Affiliation:
Laboratory of Cytokines and Antitumor Immunity, CJF 94-11 INSERM, Gustave Roussy Institute, Villejuif, France.
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MeSH Terms
Descriptor/Qualifier:
Adult
Cytotoxicity, Immunologic / drug effects*
Drug Synergism
Humans
Interferon-gamma / biosynthesis,  drug effects
Interleukin-12 / pharmacology*
Isoantigens / immunology*
Lymphocyte Activation / drug effects*
Lymphocyte Culture Test, Mixed
Receptors, Interleukin / biosynthesis,  drug effects
T-Lymphocytes, Cytotoxic / drug effects*,  metabolism
Transforming Growth Factor beta / pharmacology*
Grant Support
ID/Acronym/Agency:
AI34758/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Isoantigens; 0/Receptors, Interleukin; 0/Transforming Growth Factor beta; 187348-17-0/Interleukin-12; 82115-62-6/Interferon-gamma

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