Document Detail


Fetal programming: causes and consequences as revealed by studies of dietary manipulation in rats -- a review.
MedLine Citation:
PMID:  16533523     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
During pregnancy, the developing fetus is dependent on its mother for all nutritional requirements. It is not surprising, therefore, that variations in maternal nutrition can be reflected in alterations in fetal health and well-being. Interestingly, the changes can persist into adulthood and may result in increased risk of diseases such as diabetes, obesity and cardiovascular disease. The first observations of these phenomena resulted in the development of hypotheses collectively brought under the heading of "fetal" or, more recently, "developmental" programming. In this review, we will examine some of the animal models used to understand the mechanisms involved and attempt to determine whether there are common, "gatekeeper", pathways or genes, altered by the different nutritional stresses. We will concentrate primarily on nutrition related to post-natal development of hypertension and will restrict the review to studies in rodents, since that is where most of the mechanistic studies are being undertaken. Our conclusions are that, while there may well be some common gatekeeper pathways, there is also some diversity of mechanism which may contribute to the generation of the same or similar phenotypes.
Authors:
H J McArdle; H S Andersen; H Jones; L Gambling
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2006-03-13
Journal Detail:
Title:  Placenta     Volume:  27 Suppl A     ISSN:  0143-4004     ISO Abbreviation:  Placenta     Publication Date:  2006 Apr 
Date Detail:
Created Date:  2006-04-18     Completed Date:  2006-09-26     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  8006349     Medline TA:  Placenta     Country:  England    
Other Details:
Languages:  eng     Pagination:  S56-60     Citation Subset:  IM    
Affiliation:
Rowett Research Institute, Greenburn Road, Bucksburn, Aberdeen AB21 9SB, UK. h.mcardle@rowett.ac.uk
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MeSH Terms
Descriptor/Qualifier:
Adaptation, Physiological*
Anemia, Iron-Deficiency / diet therapy,  pathology
Animals
Diet
Diet, Fat-Restricted
Diet, Protein-Restricted
Female
Fetal Development
Fetal Growth Retardation / diet therapy,  etiology
Hypertension / etiology
Maternal Nutritional Physiological Phenomena*
Pregnancy
Pregnancy, Animal / physiology*
Prenatal Exposure Delayed Effects*
Rats

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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