Document Detail


Fetal alcohol syndrome: an assessment of the field.
MedLine Citation:
PMID:  15956764     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Fetal exposure to alcohol is the major known cause of mental retardation in the Western world. For more than half of the 20th century, the placenta was widely believed to be an effective barrier against environmental agents. The discovery that offspring of pregnant women who were exposed to German measles or administered thalidomide were often malformed raised awareness that teratogens could be any environmental agent, including viruses and drugs, that caused abnormal development. Alcohol was not identified as a teratogen until the 1970s. Fetal exposure to alcohol can cause fetal alcohol syndrome (FAS), which is characterized by specific physical traits and central nervous system dysfunctions. The development of animal model systems has facilitated our study of the effects of fetal alcohol exposure and the elucidation of the mechanisms involved in alcohol-induced abnormal development. Despite our current understanding of the effects of fetal alcohol exposure, the occurrence of FAS and associated fetal alcohol spectrum disorders is still widespread and the associated health-care costs are staggering. This symposium provides an up-to-date analysis of fetal exposure to alcohol and FAS. It is directed not only to investigators working in the field but to a diverse group of scientists working in the biological and biomedical fields to stimulate cross-disciplinary awareness, interest, and collaboration.
Authors:
James R West; Charles A Blake
Related Documents :
20145684 - Maternal alcohol use before and during pregnancy among women in taranaki, new zealand.
12554054 - Biochemical markers of alcohol use in pregnant women.
15084904 - Effect of duration of maternal alcohol consumption on calcium metabolism and bone in th...
2772574 - Occupational noise exposure and course of pregnancy.
17244754 - Pregnancy and interferon tau regulate rsad2 and ifih1 expression in the ovine uterus.
12201834 - The treatment of a thyrotropin-secreting pituitary macroadenoma with octreotide in twin...
Publication Detail:
Type:  Congresses    
Journal Detail:
Title:  Experimental biology and medicine (Maywood, N.J.)     Volume:  230     ISSN:  1535-3702     ISO Abbreviation:  Exp. Biol. Med. (Maywood)     Publication Date:  2005 Jun 
Date Detail:
Created Date:  2005-06-15     Completed Date:  2005-07-18     Revised Date:  2005-10-25    
Medline Journal Info:
Nlm Unique ID:  100973463     Medline TA:  Exp Biol Med (Maywood)     Country:  United States    
Other Details:
Languages:  eng     Pagination:  354-6     Citation Subset:  IM    
Affiliation:
Department of Human Anatomy and Medical Neurobiology, College of Medicine, The Texas A&M University System Health Science Center, College Station, Texas 77843-1114, USA. jrwest@medicine.tamhsc.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Alcohol Drinking / adverse effects*
Animals
Disease Models, Animal
Female
Fetal Alcohol Syndrome / etiology*,  prevention & control
Humans
Mental Retardation / etiology
Pregnancy
Prenatal Exposure Delayed Effects*
Teratology
Comments/Corrections
Comment In:
Exp Biol Med (Maywood). 2005 Jun;230(6):407-12   [PMID:  15956770 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Transcriptional reprogramming during reloading of atrophied rat soleus muscle.
Next Document:  Fetal alcohol spectrum disorders: an overview with emphasis on changes in brain and behavior.