Document Detail


Feasibility of D-glucuronate to enhance gamma-hydroxybutyric acid metabolism during gamma-hydroxybutyric acid toxicity: pharmacokinetic and pharmacodynamic studies.
MedLine Citation:
PMID:  17061294     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Gamma-Hydroxybutyric acid (GHB) is a drug of abuse. Literature studies showed that D-glucuronate acts as an oxidative stimulator of GHB metabolism following in vivo GHB tracer doses. The present proof-of-concept study investigates if D-glucuronate enhances GHB metabolism and inhibits blood-brain barrier (BBB) carrier-mediated transport of GHB for clinically relevant and toxicological concentrations of GHB. In a randomized cross-over study with a 3 day washout period, rats were intravenously administered GHB (200, 400 or 800 mg/kg) with either saline or D-glucuronate (830 mg/kg i.v. bolus followed by a constant infusion of 1.39 g/kg-h). Systemic and renal GHB pharmacokinetics, as well as onset, offset and duration of GHB sedative/hypnotic effects were measured following each GHB dose. In situ brain perfusion was used to determine if D-glucuronate inhibited GHB BBB transport. D-Glucuronate did not alter GHB sedative/hypnotic effects at all three GHB doses. A model independent approach revealed that GHB systemic (AUC, CL(Total), CL(Metabolism), V(SS), T(1/2)) and renal (CL(Renal), f(e)) pharmacokinetic parameters were unaltered by D-glucuronate administration. GHB influx clearance was unaltered by D-glucuronate suggesting a lack of transport inhibition. These observations suggest that although previously shown to be promising at GHB tracer doses, D-glucuronate lacks therapeutic benefit in the treatment of GHB toxicity.
Authors:
Indranil Bhattacharya; Kathleen M K Boje
Related Documents :
224974 - Behavioral and biochemical analysis of gaba-mediated inhibition in the early chick embryo.
1969494 - Activation of arginine metabolism to glutamate in rat brain synaptosomes in thioacetami...
8245784 - Identification of a dominant epitope of glutamic acid decarboxylase (gad-65) recognized...
2313934 - Acid secretagogue action of structurally gamma-aminobutyric acid (gaba)-related compoun...
1623934 - Synthesis of retinoic acid from retinol by cultured rabbit müller cells.
18980774 - The influence of disinfection on aquatic biodegradable organic carbon formation.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Biopharmaceutics & drug disposition     Volume:  28     ISSN:  0142-2782     ISO Abbreviation:  Biopharm Drug Dispos     Publication Date:  2007 Jan 
Date Detail:
Created Date:  2006-12-25     Completed Date:  2007-03-21     Revised Date:  2007-12-03    
Medline Journal Info:
Nlm Unique ID:  7911226     Medline TA:  Biopharm Drug Dispos     Country:  England    
Other Details:
Languages:  eng     Pagination:  1-11     Citation Subset:  IM    
Copyright Information:
Copyright 2006 John Wiley & Sons, Ltd.
Affiliation:
Department of Pharmaceutical Sciences, School of Pharmacy and Pharmaceutical Sciences, University at Buffalo, Buffalo, NY 14260, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Blood-Brain Barrier / drug effects,  metabolism
Glucuronates / pharmacology*
Hydroxybutyrates / pharmacokinetics*,  toxicity*
Hypnotics and Sedatives / pharmacokinetics*,  toxicity*
Kidney / drug effects,  metabolism
Male
Rats
Rats, Sprague-Dawley
Grant Support
ID/Acronym/Agency:
DA 14988/DA/NIDA NIH HHS
Chemical
Reg. No./Substance:
0/Glucuronates; 0/Hydroxybutyrates; 0/Hypnotics and Sedatives; 591-81-1/4-hydroxybutyric acid

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Implementing tandem mass spectrometry as a routine tool for characterizing the complete purine and p...
Next Document:  Effects of bosentan, an endothelin receptor antagonist, on bile salt export pump and multidrug resis...