Document Detail


FcgammaRIIa genotype predicts progression of HIV infection.
MedLine Citation:
PMID:  18025239     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Polymorphisms in FcgammaR genes are associated with susceptibility to or severity of a number of autoimmune and infectious diseases. We found that HIV-infected men in the Multicenter AIDS Cohort Study with the FcgammaRIIa RR genotype progressed to a CD4(+) cell count of <200/mm(3) at a faster rate than individuals with the RH or HH genotypes (relative hazard = 1.6; p = 0.0001). However, progression to AIDS (using the broad definition of either a CD4(+) cell count <200/mm(3) or development of an AIDS-defining illness) was less impacted by FcgammaRIIa genotype, largely because HH homozygotes had an increased risk of Pneumocystis jiroveci pneumonia as an AIDS-defining illness. We also showed that chronically infected subjects develop a substantial anti-gp120-specific IgG2 response. Moreover, HIV-1 immune complexes are more efficiently internalized by monocytes from HH subjects compared with RR subjects, likely because of the presence of IgG2 in the complexes. Finally, the FcgammaRIIIa F/V gene polymorphism was not associated with progression of HIV infection, but, as demonstrated previously, did predict the risk of Kaposi's sarcoma. These results demonstrate the importance of FcgammaRs in AIDS pathogenesis and point toward a critical role for interactions between FcgammaRs and immune complexes in disease progression.
Authors:
Donald N Forthal; Gary Landucci; Jay Bream; Lisa P Jacobson; Tran B Phan; Benjamin Montoya
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Publication Detail:
Type:  Journal Article; Multicenter Study; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of immunology (Baltimore, Md. : 1950)     Volume:  179     ISSN:  0022-1767     ISO Abbreviation:  J. Immunol.     Publication Date:  2007 Dec 
Date Detail:
Created Date:  2007-11-20     Completed Date:  2008-02-05     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  2985117R     Medline TA:  J Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  7916-23     Citation Subset:  AIM; IM    
Affiliation:
Division of Infectious Diseases, Department of Medicine, University of California, Irvine School of Medicine, Irvine, CA 92697, USA. dnfortha@uci.edu
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MeSH Terms
Descriptor/Qualifier:
Adult
Antigens, CD / genetics*
CD4 Lymphocyte Count
CD4-Positive T-Lymphocytes / immunology,  virology
Cohort Studies
Disease Progression
Genotype
HIV / immunology
HIV Infections / diagnosis*,  genetics*,  immunology
Humans
Male
Middle Aged
Predictive Value of Tests
Receptors, IgG / genetics*
Risk Factors
Grant Support
ID/Acronym/Agency:
5 M01 RR00722/RR/NCRR NIH HHS; U01 AI35039/AI/NIAID NIH HHS; U01 AI35040/AI/NIAID NIH HHS; U01 AI35041/AI/NIAID NIH HHS; U01 AI35042/AI/NIAID NIH HHS; U01 AI35043/AI/NIAID NIH HHS; U01 AI37613/AI/NIAID NIH HHS; U01 AI37984/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Antigens, CD; 0/Fc gamma receptor IIA; 0/Receptors, IgG

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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