Document Detail


Fatty acid oxidation inhibitors in the management of chronic complications of atherosclerosis.
MedLine Citation:
PMID:  15683605     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Ischemic heart disease is characterized by a modification of the normal energy balance of the heart. During and following an ischemic event, circulating fatty acids are elevated, resulting in the acceleration of fatty acid oxidation at the expense of glucose oxidation. Despite the reduction in glucose oxidation, the rate of glycolysis increases, leading to an uncoupling of glucose metabolism. This results in the accumulation of metabolic byproducts, which leads to a decrease in cardiac efficiency. A novel therapeutic strategy involves improving the efficiency of oxygen utilization by the ischemic heart by the modulation of energy metabolism. This can be achieved by a reduction in the levels of circulating fatty acids using beta-blockers, glucose-insulin-potassium infusions, and nicotinic acid. Alternatively, fatty acid oxidation can be directly inhibited using trimetazidine, ranolazine, or glucose oxidation directly activated using dichloroacetate, which significantly improves the efficiency of the heart.
Authors:
Clifford D L Folmes; Alexander S Clanachan; Gary D Lopaschuk
Related Documents :
16269705 - Abundance, activity, and community structure of pelagic methane-oxidizing bacteria in t...
7137365 - Effect of thyroxine treatment on exogenous myocardial lactate oxidation.
7763255 - Adrenoleukodystrophy: the restoration of peroxisomal beta-oxidation by transfection of ...
2565735 - Activities of enzymes of lipid metabolism in morris hepatoma 7800 c1 cells.
15177755 - Tuning the diffusion dialysis performance by surface cross-linking of ppo anion exchang...
10630125 - Fukiic and piscidic acid esters from the rhizome of cimicifuga racemosa and the in vitr...
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Current atherosclerosis reports     Volume:  7     ISSN:  1523-3804     ISO Abbreviation:  Curr Atheroscler Rep     Publication Date:  2005 Feb 
Date Detail:
Created Date:  2005-02-01     Completed Date:  2005-06-30     Revised Date:  2005-11-17    
Medline Journal Info:
Nlm Unique ID:  100897685     Medline TA:  Curr Atheroscler Rep     Country:  United States    
Other Details:
Languages:  eng     Pagination:  63-70     Citation Subset:  IM    
Affiliation:
Cardiovascular Research Group, University of Alberta, Edmonton, Alberta T6G 2S2, Canada.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Carbohydrate Metabolism
Energy Metabolism
Enzyme Inhibitors / therapeutic use
Fatty Acids / metabolism*
Humans
Myocardial Ischemia / metabolism
Myocardial Reperfusion Injury / metabolism
Myocardium / metabolism
Oxidation-Reduction / drug effects*
Chemical
Reg. No./Substance:
0/Enzyme Inhibitors; 0/Fatty Acids

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Adipocytokines: emerging therapeutic targets.
Next Document:  Antioxidants and atherosclerosis: emerging drug therapies.