| Fatty acid mobilization from adipose tissue during exercise. | |
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MedLine Citation:
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PMID: 14516937 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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By far the largest energy reserve in the human body is adipose tissue triglycerides, and these reserves are an important source of fuel during prolonged endurance exercise. To use this rich source of potential energy during exercise, adipose tissue triglycerides must first be hydrolyzed and the resultant fatty acids delivered to the working muscles. The aims of this review are to describe how exercise alters lipid mobilization from adipose tissue, to identify alternative sources of lipids and to discuss some of the key factors regulating fatty acid mobilization, uptake and oxidation during exercise. The impact of understanding factors involved in the coordinated regulation of lipid mobilization and oxidation during exercise goes far beyond its relevance for endurance exercise performance. A better understanding of the regulation of these processes will facilitate the development of more effective treatment modalities for obesity-related metabolic disorders. |
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Authors:
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Jeffrey F Horowitz |
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Publication Detail:
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Type: Journal Article; Review |
Journal Detail:
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Title: Trends in endocrinology and metabolism: TEM Volume: 14 ISSN: 1043-2760 ISO Abbreviation: Trends Endocrinol. Metab. Publication Date: 2003 Oct |
Date Detail:
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Created Date: 2003-09-30 Completed Date: 2004-07-09 Revised Date: 2005-11-16 |
Medline Journal Info:
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Nlm Unique ID: 9001516 Medline TA: Trends Endocrinol Metab Country: United States |
Other Details:
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Languages: eng Pagination: 386-92 Citation Subset: IM |
Affiliation:
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Division of Kinesiology, The University of Michigan, Ann Arbor, MI 48109-2214, USA. jeffhoro@umich.edu |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Adipose Tissue
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metabolism* Adult Energy Metabolism / physiology* Exercise / physiology* Fatty Acids / metabolism* Humans Lipolysis / physiology* |
| Chemical | |
Reg. No./Substance:
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0/Fatty Acids |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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