Document Detail


Fas determines differential fates of resident and recruited macrophages during resolution of acute lung injury.
MedLine Citation:
PMID:  21471090     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
RATIONALE: During acute lung injury (ALI) the macrophage pool expands markedly as inflammatory monocytes migrate from the circulation to the airspaces. As inflammation resolves, macrophage numbers return to preinjury levels and normal tissue structure and function are restored.
OBJECTIVES: To determine the fate of resident and recruited macrophages during the resolution of ALI in mice and to elucidate the mechanisms responsible for macrophage removal.
METHODS: ALI was induced in mice using influenza A (H1N1; PR8) infection and LPS instillation. Dye labeling techniques, bone marrow transplantation, and surface immunophenotyping were used to distinguish resident and recruited macrophages during inflammation and to study the role of Fas in determining macrophage fate during resolving ALI.
MEASUREMENTS AND MAIN RESULTS: During acute and resolving lung injury from influenza A and LPS, a high proportion of the original resident alveolar macrophages persisted. In contrast, recruited macrophages exhibited robust accumulation in early inflammation, followed by a progressive decline in their number. This decline was mediated by apoptosis with local phagocytic clearance. Recruited macrophages expressed high levels of the death receptor Fas and were rapidly depleted from the airspaces by Fas-activating antibodies. In contrast, macrophage depletion was inhibited in mice treated with Fas-blocking antibodies and in chimeras with Fas-deficient bone marrow. Caspase-8 inhibition prevented macrophage apoptosis and delayed the resolution of ALI.
CONCLUSIONS: These findings indicate that Fas-induced apoptosis of recruited macrophages is essential for complete resolution of ALI.
Authors:
William J Janssen; Lea Barthel; Alaina Muldrow; Rebecca E Oberley-Deegan; Mark T Kearns; Claudia Jakubzick; Peter M Henson
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  American journal of respiratory and critical care medicine     Volume:  184     ISSN:  1535-4970     ISO Abbreviation:  Am. J. Respir. Crit. Care Med.     Publication Date:  2011 Sep 
Date Detail:
Created Date:  2011-10-12     Completed Date:  2011-11-08     Revised Date:  2012-04-04    
Medline Journal Info:
Nlm Unique ID:  9421642     Medline TA:  Am J Respir Crit Care Med     Country:  United States    
Other Details:
Languages:  eng     Pagination:  547-60     Citation Subset:  AIM; IM    
Affiliation:
Division of Pulmonary Medicine, Department of Medicine, National Jewish Health, Denver, Colorado 80206, USA. janssenw@NJHealth.org
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MeSH Terms
Descriptor/Qualifier:
Acute Lung Injury / metabolism,  pathology*,  therapy
Animals
Antigens, CD95 / immunology*,  metabolism
Apoptosis / immunology*
Bronchoalveolar Lavage Fluid / chemistry,  cytology,  immunology
Caspase 8 / immunology
Disease Models, Animal
Flow Cytometry
Immunohistochemistry
Macrophages, Alveolar / immunology,  pathology*
Mice
Mice, Inbred C57BL
Grant Support
ID/Acronym/Agency:
GM61031/GM/NIGMS NIH HHS; HL68864/HL/NHLBI NIH HHS; HL81151/HL/NHLBI NIH HHS; R01 HL068864-09/HL/NHLBI NIH HHS; R01 HL081151-08/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Antigens, CD95; EC 3.4.22.-/Caspase 8
Comments/Corrections
Comment In:
Am J Respir Crit Care Med. 2011 Sep 1;184(5):497-8   [PMID:  21885631 ]

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