Document Detail


FOXD1 Duplication Causes Branchial Defects and Interacts with the TFAP2A Gene Implicated in the Branchio-Oculo-Facial Syndrome in Causing Eye Effects in Zebrafish.
MedLine Citation:
PMID:  22140378     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
Branchio-oculo-facial syndrome (BOFS) is a rare disorder characterized by maldevelopment of the first and second branchial arches, skin defects, facial dysmorphism, auricular, ophthalmological and oral abnormalities. A high clinical variability has been reported. Recently, mutations in TFAP2A were found to underlie this condition. A small duplication on 5q13 was detected in 2 family members with mild BOFS features. Molecular cytogenetic delineation of the duplication demonstrated that only 7 genes are affected: LOC100289045, RGNEF, UTP15, ANKRA2, FUNDC2P1, BTF3 and FOXD1. The latter is expressed in the developing branchial arches and involved in cranio-facial development. Zebrafish embryos with combined inhibition of the expression of foxd1l and tfap2a show optic axis defects. We identified a novel locus associated with a mild BOFS-like phenotype. The functional in vivo experiments suggest an interaction between FOXD1 and TFAP2A.
Authors:
I Balikova; K Devriendt; J-P Fryns; J R Vermeesch
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Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2011-5-18
Journal Detail:
Title:  Molecular syndromology     Volume:  1     ISSN:  1661-8777     ISO Abbreviation:  -     Publication Date:  2010  
Date Detail:
Created Date:  2011-12-5     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101525192     Medline TA:  Mol Syndromol     Country:  -    
Other Details:
Languages:  ENG     Pagination:  255-261     Citation Subset:  -    
Affiliation:
Center for Human Genetics, Catholic University Leuven, Leuven, Belgium.
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