Document Detail


Expression of laminin-2 by normal and neoplastic rat C cells during the development of medullary thyroid carcinoma.
MedLine Citation:
PMID:  10335943     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Medullary thyroid carcinoma (MTC) originates from C cells, which secrete calcitonin (CT), their specific marker. C cells are located in contact with the basement membrane (BM) of the thyroid follicles, which is partly made up of the laminin-2 isoform synthesized by thyrocytes. During oncogenesis, proliferation of the C cells, invading the centre of the follicles, leads to a break in their normal contact with the BM. As specific interactions of cells with BM components, especially laminins, are important for proliferation and differentiation, we investigated the relationships of normal and neoplastic C cells with laminin in the Wag/Rij rat model of human MTC. Immunocytochemical studies showed a progressive loss of the laminin layer underlying the hyperplastic C cell nodules around the large dedifferentiated tumours. The alpha2, beta1 and gamma1 chains of the laminin-2 isoform were synthesized and secreted by rat MTC 6-23 cell cultures and the tumours induced by subcutaneous injection of these cells. In situ hybridization combined with anti-CT immunocytochemistry showed a low expression of alpha2 mRNA on differentiated C cells and thyrocytes, but an overexpression on immunonegative spontaneous MTC and induced intrathyroid tumours. The high level of alpha2 gene expression, together with tumour dedifferentiation, suggests a relationship with malignancy.
Authors:
F Lekmine; H Feracci; G Milhaud; F Treilhou-Lahille; N Jeanne
Related Documents :
6261443 - Immunohistochemical and ultrastructural studies on rat islet cell tumours induced by st...
2133463 - Feasibility of the immunohistochemical detection of endogenous steroids in paraffin-emb...
6601203 - Tissue factor activity of normal and neoplastic cells: quantitation and species specifi...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Virchows Archiv : an international journal of pathology     Volume:  434     ISSN:  0945-6317     ISO Abbreviation:  Virchows Arch.     Publication Date:  1999 Apr 
Date Detail:
Created Date:  1999-06-03     Completed Date:  1999-06-03     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  9423843     Medline TA:  Virchows Arch     Country:  GERMANY    
Other Details:
Languages:  eng     Pagination:  325-32     Citation Subset:  IM    
Affiliation:
Laboratoire d'Endocrinologie Cellulaire et Evolution, URA 1116 CNRS, Université Paris-Sud, Orsay, France.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Basement Membrane / metabolism,  pathology
Carcinoma, Medullary / metabolism*,  pathology
DNA Primers / chemistry
Disease Models, Animal
Female
In Situ Hybridization
Laminin / biosynthesis*,  genetics
Male
Neoplasm Transplantation
Neoplasms, Experimental / metabolism,  pathology
RNA, Messenger / biosynthesis
Rats
Rats, Inbred Strains
Reverse Transcriptase Polymerase Chain Reaction
Thyroid Gland / metabolism*,  pathology
Thyroid Neoplasms / metabolism*,  pathology
Tumor Cells, Cultured / metabolism,  pathology
Chemical
Reg. No./Substance:
0/DNA Primers; 0/Laminin; 0/RNA, Messenger; 0/laminin alpha 2

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Lymphoepithelial duct lesions in Sjögren-type sialadenitis.
Next Document:  The interaction of Bcl-2 and Bax regulates apoptosis in biliary epithelial cells of rats with obstru...