Document Detail


Exposure to ultrafine particles in asphalt work.
MedLine Citation:
PMID:  18850455     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
An epidemiologic study has demonstrated that asphalt workers show increased loss of lung function and an increase of biomarkers of inflammation over the asphalt paving season. The aim of this study was to investigate which possible agent(s) causes the inflammatory reaction, with emphasis on ultrafine particles. The workers' exposure to total dust, polycyclic aromatic hydrocarbons, and NO(2) was determined by personal sampling. Exposure to ultrafine particles was measured by means of particle counters and scanning mobility particle sizer mounted on a van following the paving machine. The fractions of organic and elemental carbon were determined. Asphalt paving workers were exposed to ultrafine particles with medium concentration of about 3.4 x 10(4)/cm(3). Ultrafine particles at the paving site originated mainly from asphalt paving activities and traffic exhaust; most seemed to originate from asphalt fumes. Oil mist exceeded occupational limits on some occasions. Diesel particulate matter was measured as elemental carbon, which was low, around 3 microg/m(3). NO(2) and total dust did not exceed limits. Asphalt pavers were exposed to relatively high concentrations of ultrafine particles throughout their working day, with possible adverse health effects.
Authors:
Karine Elihn; Bente Ulvestad; Siri Hetland; Anna Wallen; Britt Grethe Randem
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of occupational and environmental hygiene     Volume:  5     ISSN:  1545-9632     ISO Abbreviation:  J Occup Environ Hyg     Publication Date:  2008 Dec 
Date Detail:
Created Date:  2008-10-13     Completed Date:  2008-11-14     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101189458     Medline TA:  J Occup Environ Hyg     Country:  United States    
Other Details:
Languages:  eng     Pagination:  771-9     Citation Subset:  IM    
Affiliation:
Department of Applied Environmental Science, Stockholm University, Stockholm, Sweden. Karine.Elihn@itm.su.se
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MeSH Terms
Descriptor/Qualifier:
Carbon / analysis,  toxicity
Dust / analysis
Environmental Monitoring*
Humans
Hydrocarbons / analysis,  chemistry,  toxicity*
Lung Diseases / chemically induced,  etiology*,  pathology
Nitrogen Oxides / analysis,  toxicity
Occupational Exposure / analysis*
Particle Size
Polycyclic Hydrocarbons, Aromatic / analysis,  toxicity
Vehicle Emissions / analysis,  toxicity
Chemical
Reg. No./Substance:
0/Dust; 0/Hydrocarbons; 0/Nitrogen Oxides; 0/Polycyclic Hydrocarbons, Aromatic; 0/Vehicle Emissions; 7440-44-0/Carbon; 8052-42-4/asphalt

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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