Document Detail


Exposure to bioaccumulative organochlorine compounds alters adipogenesis, fatty acid uptake, and adipokine production in NIH3T3-L1 cells.
MedLine Citation:
PMID:  21044676     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Exposure to the organochlorine compounds p,p'-dichlorodiphenyldichloroethylene (DDE) and oxychlordane have been associated with an increased prevalence of diabetes. Although the exact etiology of diabetes, especially type 2 diabetes, is not known, it is thought that adipose dysfunction plays a vital role in the progression of this disease. Thus, the present study examined whether exposure to these bioaccumulative compounds promotes adipocyte dysfunction including alterations in adipogenesis, fatty acid storage, and adipokine production within the adipocyte. We employed the NIH3T3-L1 cell line as a model for adipogenesis and mature adipocyte function. Exposure to DDE or oxychlordane prior to and throughout differentiation did not affect adipogenesis. In mature NIH3T3-L1 adipocytes, exposure to oxychlordane, DDE, or dieldrin had no effect on insulin-stimulated fatty acid uptake but did increase basal fatty acid uptake over a 24 h period. There was no observed effect of exposure to these compounds on lipolysis. Exposure to DDE significantly increased the release of leptin, resistin, and adiponectin from mature adipocytes with corresponding increases in expression of resistin and adiponectin. Taken together, the current data suggest that exposure to these compounds, especially DDE, may promote some aspects of adipocyte dysfunction that are commonly associated with obesity and type 2 diabetes.
Authors:
George Howell; Lauren Mangum
Related Documents :
11751846 - Insulin acutely regulates munc18-c subcellular trafficking: altered response in insulin...
16154996 - Characterization of the role of the rab gtpase-activating protein as160 in insulin-regu...
14972746 - Evidence that nitroprusside stimulates glucose uptake in isolated rat cardiomyocytes vi...
20363216 - Insulinotropic effect of cinnamaldehyde on transcriptional regulation of pyruvate kinas...
1828656 - Insulin plus a sulfonylurea agent for treating type 2 diabetes.
19654286 - Inhibition of hepatic niemann-pick c1-like 1 improves hepatic insulin resistance.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-10-31
Journal Detail:
Title:  Toxicology in vitro : an international journal published in association with BIBRA     Volume:  25     ISSN:  1879-3177     ISO Abbreviation:  Toxicol In Vitro     Publication Date:  2011 Feb 
Date Detail:
Created Date:  2010-12-28     Completed Date:  2011-05-17     Revised Date:  2012-04-09    
Medline Journal Info:
Nlm Unique ID:  8712158     Medline TA:  Toxicol In Vitro     Country:  England    
Other Details:
Languages:  eng     Pagination:  394-402     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Ltd. All rights reserved.
Affiliation:
Center for Environmental Health Sciences, College of Veterinary Medicine, Mississippi State University, Mississippi State, MS 39762, USA. howell@cvm.msstate.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adipocytes, White / drug effects*,  metabolism*
Adipogenesis / drug effects*
Adipokines / genetics,  metabolism*
Adiponectin / genetics,  metabolism
Animals
Biological Transport / drug effects
Chlordan / analogs & derivatives,  toxicity
Dichlorodiphenyl Dichloroethylene / toxicity
Dieldrin / toxicity
Fatty Acids / metabolism*
Gene Expression Regulation / drug effects
Hydrocarbons, Chlorinated / toxicity*
Insulin / metabolism
Leptin / genetics,  metabolism
Lipolysis / drug effects
Mice
NIH 3T3 Cells
Pesticides / toxicity*
RNA, Messenger / metabolism
Resistin / genetics,  metabolism
Grant Support
ID/Acronym/Agency:
1K01ES019182/ES/NIEHS NIH HHS; 1P20-RR17661/RR/NCRR NIH HHS; 1R15ES019742/ES/NIEHS NIH HHS; K01 ES019182-01/ES/NIEHS NIH HHS; K01 ES019182-02/ES/NIEHS NIH HHS
Chemical
Reg. No./Substance:
0/Adipokines; 0/Adiponectin; 0/Adipoq protein, mouse; 0/Fatty Acids; 0/Hydrocarbons, Chlorinated; 0/Insulin; 0/Leptin; 0/Pesticides; 0/RNA, Messenger; 0/Resistin; 0/Retn protein, mouse; 12789-03-6/Chlordan; 27304-13-8/oxychlordane; 60-57-1/Dieldrin; 72-55-9/Dichlorodiphenyl Dichloroethylene
Comments/Corrections
Erratum In:
Toxicol In Vitro. 2011 Oct;25(7):1507

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  A review on biological, nutraceutical and clinical aspects of French maritime pine bark extract.
Next Document:  Encapsulation of cadmium selenide quantum dots using a self-assembling nanoemulsion (SANE) reduces t...