Document Detail


Exercise training decreases hepatic SCD-1 gene expression and protein content in rats.
MedLine Citation:
PMID:  19777326     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Stearoyl-CoA desaturase-1 (SCD-1) is the rate limiting enzyme in the biosynthesis of saturated-derived monounsaturated fats that are the major constituents of very-low-density-lipoproteins-triacylglycerol (VLDL-TAG) and are involved in regulating cellular metabolism. The purpose of this study was to evaluate the effects of an 8-week exercise training program on the hepatic gene expression of this crucial enzyme. Female rats either trained (TR) or kept sedentary (Sed) for 8 weeks were submitted either to standard (SD) diet for 8 or for 6 weeks followed by high-fat (HF; 42% kcal) diet for 2 weeks. The 2-week-high fat feeding resulted in an increase in liver triacylgycerol (TAG), plasma free-fatty-acids (FFA), abdominal fat mass, sterol-regulatory-element-binding protein-1c (SREBP-1c), and carbohydrate-response-element-binding protein (ChREBP) gene expression in liver along with a decrease in SCD-1 gene expression and plasma and liver SCD-1 desaturation index (C16:1/C16:0). Liver TAG, plasma FFA, SREBP-1c mRNA, and SCD-1 desaturation indexes (C16:1/C16:0; C18:1/C18:0) were not changed in liver or in plasma by the training program. Nevertheless, training resulted in an important decrease in fat mass (P < 0.01), hepatic SCD-1 mRNA levels (P < 0.01), and protein content (P < 0.05) in both SD and HF fed rats. It is concluded that despite an absence of decreased liver TAG, exercise training contributes to the proper regulation of fat metabolism by down-regulating hepatic SCD-1 gene expression and protein content.
Authors:
Siham Yasari; Denis Prud'homme; Donghao Wang; Marek Jankowski; Emile Levy; Jolanta Gutkowska; Jean-Marc Lavoie
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-09-24
Journal Detail:
Title:  Molecular and cellular biochemistry     Volume:  335     ISSN:  1573-4919     ISO Abbreviation:  Mol. Cell. Biochem.     Publication Date:  2010 Feb 
Date Detail:
Created Date:  2010-01-21     Completed Date:  2010-05-13     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0364456     Medline TA:  Mol Cell Biochem     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  291-9     Citation Subset:  IM    
Affiliation:
Department of Kinesiology, University of Montreal, Montreal, Canada. syasari@uottawa.ca
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MeSH Terms
Descriptor/Qualifier:
Animals
Basic Helix-Loop-Helix Leucine Zipper Transcription Factors / genetics,  metabolism
Dietary Fats / metabolism
Fatty Acids, Nonesterified / metabolism
Female
Gene Expression
Lipid Metabolism
Liver / enzymology*,  metabolism
Physical Conditioning, Animal*
RNA, Messenger / metabolism
Rats
Rats, Sprague-Dawley
Stearoyl-CoA Desaturase / genetics*,  metabolism
Sterol Regulatory Element Binding Protein 1 / genetics,  metabolism
Triglycerides / metabolism
Grant Support
ID/Acronym/Agency:
MOP-10584//Canadian Institutes of Health Research; MOP-49433//Canadian Institutes of Health Research; MOP-53217//Canadian Institutes of Health Research; MOP-62901//Canadian Institutes of Health Research; T0602145.02//Canadian Institutes of Health Research
Chemical
Reg. No./Substance:
0/Basic Helix-Loop-Helix Leucine Zipper Transcription Factors; 0/Dietary Fats; 0/Fatty Acids, Nonesterified; 0/RNA, Messenger; 0/Sterol Regulatory Element Binding Protein 1; 0/Triglycerides; 0/Wbscr14 protein, rat; EC 1.14.19.1/Stearoyl-CoA Desaturase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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