| Exercise as a mediator of hepcidin activity in athletes. | |
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MedLine Citation:
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PMID: 20697906 Owner: NLM Status: In-Process |
Abstract/OtherAbstract:
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Iron is a trace mineral used by the body in many physiological processes that are essential for athletic performance. However, it is common that an athlete's iron stores are compromised via several well-established exercise-related mechanisms such as hemolysis, hematuria, sweating and gastrointestinal bleeding. Recently, however, a new mechanism for athletics-induced iron deficiency has been proposed, involving the influence of physical activity on the post-exercise hepcidin response. Hepcidin is a liver-produced hormone that regulates iron metabolism in the gut and macrophages. This hormone has become the focus of recent investigations into altered iron metabolism in athletes, and may be a mitigating factor implicated in athletics-induced iron deficiency. This review attempts to summarize and disseminate the collective knowledge currently held regarding exercise and hepcidin expression, in addition to suggesting the direction for future research in this area. |
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Authors:
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Peter Peeling |
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Publication Detail:
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Type: Journal Article Date: 2010-08-10 |
Journal Detail:
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Title: European journal of applied physiology Volume: 110 ISSN: 1439-6327 ISO Abbreviation: Eur. J. Appl. Physiol. Publication Date: 2010 Nov |
Date Detail:
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Created Date: 2010-11-05 Completed Date: - Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 100954790 Medline TA: Eur J Appl Physiol Country: Germany |
Other Details:
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Languages: eng Pagination: 877-83 Citation Subset: IM |
Affiliation:
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School of Sport Science, Exercise and Health, The University of Western Australia, Crawley, Australia. ppeeling@wais.org.au |
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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