Document Detail


Evidence for operation of the direct zinc ligand exchange mechanism for trafficking, transport, and reactivity of zinc in mammalian cells.
MedLine Citation:
PMID:  21440525     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In addition to its critical role in normal cell function, growth, and metabolism, zinc is implicated as a major factor in the development and progression of many pathological conditions and diseases. Despite this importance of zinc, many important factors, processes, and mechanisms of the physiology, biochemistry, and molecular biology of zinc remain unknown. Especially important is the unresolved issue regarding the mechanism and process of the trafficking, transport, and reactivity of zinc in cells; especially in mammalian cells. This presentation focuses on the concept that, due to the existence of a negligible pool of free Zn(2+) ions in the mammalian cell environment, the trafficking, transport and reactivity of zinc occurs via a direct exchange of zinc from donor Zn-ligands to acceptor ligands. This Zn exchange process occurs without the requirement for production of free Zn(2+) ions. The direct evidence from mammalian cell studies is presented in support of the operation of the direct Zn-ligand exchange mechanism. The paper also provides important information and conditions that should be considered and employed in the conduct of studies regarding the role and effects of zinc in biological/biomedical research; and in its clinical interpretation and application.
Authors:
Leslie C Costello; Catherine C Fenselau; Renty B Franklin
Related Documents :
1011055 - Selective release of lysosomal enzymes from cell populations containing multinucleate g...
911685 - Cutaneous infection by cryptococcus laurentii.
21262815 - Interspecies pheromone signaling promotes biofilm formation and same-sex mating in cand...
1755105 - Atypical tumor of the mediastinum: epithelioid hemangioendothelioma containing metaplas...
11536995 - The role of cellulases in the mechanism of changes of cell walls of funaria hygrometric...
21368295 - The btb-kelch protein krp1 regulates proliferation and differentiation of myoblasts.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review     Date:  2011-03-29
Journal Detail:
Title:  Journal of inorganic biochemistry     Volume:  105     ISSN:  1873-3344     ISO Abbreviation:  J. Inorg. Biochem.     Publication Date:  2011 May 
Date Detail:
Created Date:  2011-04-25     Completed Date:  2011-08-26     Revised Date:  2012-05-02    
Medline Journal Info:
Nlm Unique ID:  7905788     Medline TA:  J Inorg Biochem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  589-99     Citation Subset:  IM    
Copyright Information:
Copyright © 2011 Elsevier Inc. All rights reserved.
Affiliation:
Department of Oncology and Diagnostic Sciences, Dental School, University of Maryland, Baltimore, MD 21201, USA. lcostello@umaryland.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Biological Transport
Cell Membrane / metabolism
Cytosol / metabolism
Humans
Kinetics
Ligands
Mammals / metabolism
Mitochondria / metabolism
Models, Biological
Zinc / metabolism*
Grant Support
ID/Acronym/Agency:
R01 CA071207-08/CA/NCI NIH HHS; R01 CA079903-09/CA/NCI NIH HHS; R01 DK042839-12/DK/NIDDK NIH HHS; R01CA79903/CA/NCI NIH HHS; R01DK42839/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Ligands; 7440-66-6/Zinc

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Vitamin D and the vitamin D receptor in liver pathophysiology.
Next Document:  Single, restriction enzyme-assisted megaprimer PCR to fuse two DNA sequences on separate cloning vec...