Document Detail


Erythropoietin can promote survival of cerebral cells by downregulating Bax gene after traumatic brain injury in rats.
MedLine Citation:
PMID:  20139499     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: Traumatic brain injury (TBI) is an important cause of adult mortality and morbidity. Erythropoietin (Epo) has been shown to promote the viability of cerebral cells by upregulating Bcl-2 gene; however, Epo may exert its antiapoptotic effect via the differential regulation of the expression of genes involved in the apoptotic process. AIM: The present study examined the neuroprotective effect of Epo as a survival factor through the regulation of the Bax. MATERIALS AND METHODS: Wistar rats were randomly divided into three groups: Recombinant human EPO treated (rhEPO) TBI, vehicle-treated TBI, and sham-operated. Traumatic brain injury was induced by the Feeney free-falling model. Rats were killed 5, 12, 24, 72, 120, or 168 h after TBI. Regulation of Bcl-2 was detected by reverse transcription-polymerase chain reaction (RT-PCR), western blotting and immunofluorescence. RESULTS: Bax mRNA and protein levels were lower in the rhEPO)-treated rat brains than in the vehicle-treated rat brains. Induction of Bax expression peaked at 24 h and remained stable for 72-120 h in vehicle-treated rat brains, whereas induction of Bax expression was only slightly elevated in rhEPO-treated rat brains. The number of TdT-mediated dUTP Nick-End Labeling(TUNEL)-positive cells in the rhEPO-treated rat brains was far fewer than in the vehicle-treated rat brains. CONCLUSIONS: Epo exerts neuroprotective effect against traumatic brain injury via reducing Bax gene expression involved in inhibiting TBI-induced neuronal cell death.
Authors:
Z B Liao; G Y Jiang; Z H Tang; X G Zhi; X C Sun; W Y Tang; M J Wu
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Neurology India     Volume:  57     ISSN:  0028-3886     ISO Abbreviation:  Neurol India     Publication Date:    2009 Nov-Dec
Date Detail:
Created Date:  2010-02-08     Completed Date:  2010-04-22     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0042005     Medline TA:  Neurol India     Country:  India    
Other Details:
Languages:  eng     Pagination:  722-8     Citation Subset:  IM    
Affiliation:
Department of Neurosurgery, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China. liaozhengbu@hotmail.com
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MeSH Terms
Descriptor/Qualifier:
Analysis of Variance
Animals
Brain Injuries* / drug therapy,  pathology,  physiopathology
Cell Survival / drug effects
Cerebral Cortex / drug effects,  pathology*
Disease Models, Animal
Down-Regulation / drug effects*
Erythropoietin / pharmacology*,  therapeutic use*
Humans
In Situ Nick-End Labeling / methods
RNA, Messenger / metabolism
Rats
Rats, Wistar
Time Factors
bcl-2-Associated X Protein / genetics,  metabolism*
Chemical
Reg. No./Substance:
0/RNA, Messenger; 0/bcl-2-Associated X Protein; 11096-26-7/Erythropoietin
Comments/Corrections
Comment In:
Neurol India. 2009 Nov-Dec;57(6):693-4   [PMID:  20139494 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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