Document Detail


Enhancement of biliary phospholipid secretion by diethyl maleate administration in the rabbit.
MedLine Citation:
PMID:  3446199     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The effect of diethyl maleate administration on biliary phospholipid secretion was studied in male New Zealand rabbits. Bile flow was significantly enhanced during the first 40 min following i.p. injection of diethyl maleate at 3.2 mmol/kg body wt., after which it decreased progressively to values half those of the controls at 2 h of injection of the chemical. Bile acid secretion was not significantly modified during choleresis but was significantly reduced from 60 min of diethyl maleate administration onwards. A significant increase in biliary phospholipid secretion parallel to that occurring in bile flow was observed in diethyl maleate-treated rabbits. Our data confirm the uncoupling previously described for the rat between bile acid and phospholipid secretion into bile during diethyl maleate-induced choleresis.
Authors:
J Gonzalez; R Jimenez; A Esteller
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Biomedica biochimica acta     Volume:  46     ISSN:  0232-766X     ISO Abbreviation:  Biomed. Biochim. Acta     Publication Date:  1987  
Date Detail:
Created Date:  1988-05-05     Completed Date:  1988-05-05     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  8304435     Medline TA:  Biomed Biochim Acta     Country:  GERMANY, EAST    
Other Details:
Languages:  eng     Pagination:  713-7     Citation Subset:  IM    
Affiliation:
Department of Physiology and Pharmacology, Faculty of Pharmacy, University of Salamanca, Spain.
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MeSH Terms
Descriptor/Qualifier:
Animals
Bile / drug effects*,  secretion
Bile Acids and Salts / secretion
Cholagogues and Choleretics / pharmacology
Male
Maleates / pharmacology*
Phospholipids / secretion*
Rabbits
Chemical
Reg. No./Substance:
0/Bile Acids and Salts; 0/Cholagogues and Choleretics; 0/Maleates; 0/Phospholipids; 141-05-9/diethyl maleate

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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