Document Detail


Endogenous prostaglandin formation in the field-stimulated guinea-pig vas deferens: comparison of the inhibitory effects of indomethacin and NS-398.
MedLine Citation:
PMID:  10718109     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Prostaglandin (PG) E2 inhibited both phases of contraction produced by electrical field stimulation of the guinea-pig vas deferens. PGF2alpha and PGD2 were without effect on this preparation. Carbacyclin (a PGI2) analogue inhibited the first phase of contraction at higher concentrations, whereas U46619 (a thromboxane mimetic) potentiated both phases of contraction. As exogenous arachidonic acid inhibits both phases of contraction of the electrically field-stimulated guinea-pig vas deferens, it is likely that the arachidonic acid is converted to PGE2 in the vas deferens. Indomethacin, a non-specific inhibitor of prostaglandin H synthase (PGHS), attenuated the inhibitory actions of exogenous arachidonic acid when examined on the first phase of contraction. NS-398, a relatively specific inhibitor of PGHS-2, also prevented the inhibitory action of exogenous arachidonic acid. However, NS-398 was much less effective than indomethacin in this respect even though NS-398 and indomethacin inhibit PGHS-2 with similar potencies. Consequently, the findings suggest that exogenous arachidonic acid is converted to PGE2 in the guinea-pig vas deferens by the actions of PGHS-1 and, to a lesser extent, by PGHS-2.
Authors:
M R Miller; F C Johnson; N L Poyser
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Publication Detail:
Type:  Comparative Study; Journal Article    
Journal Detail:
Title:  Prostaglandins, leukotrienes, and essential fatty acids     Volume:  61     ISSN:  0952-3278     ISO Abbreviation:  Prostaglandins Leukot. Essent. Fatty Acids     Publication Date:  1999 Dec 
Date Detail:
Created Date:  2000-04-03     Completed Date:  2000-04-03     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  8802730     Medline TA:  Prostaglandins Leukot Essent Fatty Acids     Country:  SCOTLAND    
Other Details:
Languages:  eng     Pagination:  363-8     Citation Subset:  IM    
Affiliation:
Department of Pharmacology, University of Edinburgh Medical School, UK.
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MeSH Terms
Descriptor/Qualifier:
Animals
Arachidonic Acid / metabolism
Cyclooxygenase Inhibitors / pharmacology
Dinoprostone / metabolism
Electric Stimulation
Guinea Pigs
Indomethacin / pharmacology
Isoenzymes / drug effects
Male
Muscle Contraction / drug effects
Nitrobenzenes / pharmacology
Prostaglandin-Endoperoxide Synthases / drug effects*
Prostaglandins / biosynthesis*
Sulfonamides / pharmacology
Vas Deferens / metabolism*
Chemical
Reg. No./Substance:
0/Cyclooxygenase Inhibitors; 0/Isoenzymes; 0/Nitrobenzenes; 0/Prostaglandins; 0/Sulfonamides; 123653-11-2/N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide; 363-24-6/Dinoprostone; 506-32-1/Arachidonic Acid; 53-86-1/Indomethacin; EC 1.14.99.1/Prostaglandin-Endoperoxide Synthases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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